Document Detail


The Yin and Yang of oxidation in the development of the fatty streak. A review based on the 1994 George Lyman Duff Memorial Lecture.
MedLine Citation:
PMID:  8673557     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Recent data support the hypothesis that the fatty streak develops in response to specific phospholipids contained in LDL that become trapped in the artery wall and become oxidized as a result of exposure to the oxidative waste of the artery wall cells. The antioxidants present within both LDL and the microenvironments in which LDL is trapped function to prevent the formation of these biologically active, oxidized lipids. Enzymes associated with LDL and HDL (eg, platelet activating factor acetylhydrolase) or with HDL alone (eg, paraoxonase) destroy these biologically active lipids. The regulation and expression of these enzymes are determined genetically and are also significantly modified by environmental influences, including the acute-phase response or an atherogenic diet. The balance of these multiple factors leads to an induction or suppression of the inflammatory response in the artery wall and determines the clinical course.
Authors:
M Navab; J A Berliner; A D Watson; S Y Hama; M C Territo; A J Lusis; D M Shih; B J Van Lenten; J S Frank; L L Demer; P A Edwards; A M Fogelman
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.; Review    
Journal Detail:
Title:  Arteriosclerosis, thrombosis, and vascular biology     Volume:  16     ISSN:  1079-5642     ISO Abbreviation:  Arterioscler. Thromb. Vasc. Biol.     Publication Date:  1996 Jul 
Date Detail:
Created Date:  1996-08-15     Completed Date:  1996-08-15     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  9505803     Medline TA:  Arterioscler Thromb Vasc Biol     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  831-42     Citation Subset:  IM    
Affiliation:
Atherosclerosis Research Unit, School of Medicine, University of California at Los Angeles 90095-1736, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Arteries / metabolism
Arteriosclerosis / etiology*,  genetics
Humans
Lipid Peroxidation*
Lipoproteins, HDL / metabolism
Lipoproteins, LDL / metabolism
Phospholipids / metabolism
Grant Support
ID/Acronym/Agency:
HL 30568/HL/NHLBI NIH HHS; RR-865/RR/NCRR NIH HHS
Chemical
Reg. No./Substance:
0/Lipoproteins, HDL; 0/Lipoproteins, LDL; 0/Phospholipids

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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