Document Detail


Yeast IME2 functions early in meiosis upstream of cell cycle-regulated SBF and MBF targets.
MedLine Citation:
PMID:  22393365     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
BACKGROUND: In Saccharomyces cerevisiae, the G1 cyclin/cyclin-dependent kinase (CDK) complexes Cln1,-2,-3/Cdk1 promote S phase entry during the mitotic cell cycle but do not function during meiosis. It has been proposed that the meiosis-specific protein kinase Ime2, which is required for normal timing of pre-meiotic DNA replication, is equivalent to Cln1,-2/Cdk1. These two CDK complexes directly catalyze phosphorylation of the B-type cyclin/CDK inhibitor Sic1 during the cell cycle to enable its destruction. As a result, Clb5,-6/Cdk1 become activated and facilitate initiation of DNA replication. While Ime2 is required for Sic1 destruction during meiosis, evidence now suggests that Ime2 does not directly catalyze Sic1 phosphorylation to target it for destabilization as Cln1,-2/Cdk1 do during the cell cycle.
METHODOLOGY/PRINCIPAL FINDINGS: We demonstrated that Sic1 is eventually degraded in meiotic cells lacking the IME2 gene (ime2Δ), supporting an indirect role of Ime2 in Sic1 destruction. We further examined global RNA expression comparing wild type and ime2Δ cells. Analysis of these expression data has provided evidence that Ime2 is required early in meiosis for normal transcription of many genes that are also periodically expressed during late G1 of the cell cycle.
CONCLUSIONS/SIGNIFICANCE: Our results place Ime2 at a position in the early meiotic pathway that lies upstream of the position occupied by Cln1,-2/Cdk1 in the analogous cell cycle pathway. Thus, Ime2 may functionally resemble Cln3/Cdk1 in promoting S phase entry, or it could play a role even further upstream in the corresponding meiotic cascade.
Authors:
George S Brush; Nicole A Najor; Alan A Dombkowski; Daniela Cukovic; Kara E Sawarynski
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2012-02-29
Journal Detail:
Title:  PloS one     Volume:  7     ISSN:  1932-6203     ISO Abbreviation:  PLoS ONE     Publication Date:  2012  
Date Detail:
Created Date:  2012-03-06     Completed Date:  2012-07-23     Revised Date:  2013-05-20    
Medline Journal Info:
Nlm Unique ID:  101285081     Medline TA:  PLoS One     Country:  United States    
Other Details:
Languages:  eng     Pagination:  e31575     Citation Subset:  IM    
Affiliation:
Department of Oncology, Wayne State University School of Medicine, Detroit, Michigan, United States of America. brushg@karmanos.org
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MeSH Terms
Descriptor/Qualifier:
Amino Acid Motifs
CDC2 Protein Kinase / metabolism
Catalysis
Cell Cycle
Cyclin-Dependent Kinase Inhibitor Proteins / metabolism
Cyclin-Dependent Kinases / metabolism*
Cyclins / metabolism
DNA Replication
Epistasis, Genetic
Gene Expression Profiling
Intracellular Signaling Peptides and Proteins / genetics,  physiology*
Meiosis
Models, Biological
Models, Genetic
Oligonucleotide Array Sequence Analysis
Phosphorylation
Ploidies
Protein-Serine-Threonine Kinases / genetics,  physiology*
Repressor Proteins / metabolism
Saccharomyces cerevisiae / metabolism*,  physiology
Saccharomyces cerevisiae Proteins / genetics,  metabolism,  physiology*
Grant Support
ID/Acronym/Agency:
P30CA22453/CA/NCI NIH HHS; T32CA009531/CA/NCI NIH HHS
Chemical
Reg. No./Substance:
0/CLN1 protein, S cerevisiae; 0/CLN2 protein, S cerevisiae; 0/Cyclin-Dependent Kinase Inhibitor Proteins; 0/Cyclins; 0/Intracellular Signaling Peptides and Proteins; 0/Repressor Proteins; 0/SIC1 protein, S cerevisiae; 0/Saccharomyces cerevisiae Proteins; 0/Whi5 protein, S cerevisiae; EC 2.7.11.1/IME2 protein, S cerevisiae; EC 2.7.11.1/Protein-Serine-Threonine Kinases; EC 2.7.11.22/CDC2 Protein Kinase; EC 2.7.11.22/Cyclin-Dependent Kinases
Comments/Corrections

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