Document Detail


XRCC1 phosphorylation by CK2 is required for its stability and efficient DNA repair.
MedLine Citation:
PMID:  20471329     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
XRCC1 is a scaffold protein that interacts with several DNA repair proteins and plays a critical role in DNA base excision repair (BER). XRCC1 protein is in a tight complex with DNA ligase IIIalpha (Lig III) and this complex is involved in the ligation step of both BER and repair of DNA single strand breaks. The majority of XRCC1 has previously been demonstrated to exist in a phosphorylated form and cells containing mutant XRCC1, that is unable to be phosphorylated, display a reduced rate of single strand break repair. Here, in an unbiased assay, we demonstrate that the cytoplasmic form of the casein kinase 2 (CK2) protein is the major protein kinase activity involved in phosphorylation of XRCC1 in human cell extracts and that XRCC1 phosphorylation is required for XRCC1-Lig III complex stability. We demonstrate that XRCC1-Lig III complex containing mutant XRCC1, in which CK2 phosphorylation sites have been mutated, is unstable. We also find that a knockdown of CK2 by siRNA results in both reduced XRCC1 phosphorylation and stability, which also leads to a reduced amount of Lig III and accumulation of DNA strand breaks. We therefore propose that CK2 plays an important role in DNA repair by contributing to the stability of XRCC1-Lig III complex.
Authors:
Jason L Parsons; Irina I Dianova; David Finch; Phillip S Tait; Cecilia E Ström; Thomas Helleday; Grigory L Dianov
Related Documents :
11027979 - Dna repair mechanisms and acute myeloblastic leukemia.
8352959 - Dna repair.
18359739 - Nucleotide excision repair deficiencies and the somatotropic axis in aging.
17432829 - Lesion specificity in the base excision repair enzyme hneil1: modeling and dynamics stu...
9594429 - 2-deoxy-d-glucose induced modulation of dna damage repair, survival, mutagenesis and re...
18083609 - Mechanism of oxidative dna damage repair and relevance to human pathology.
19879979 - Regulatory mechanism of the initiation step of dna replication by cdk in budding yeast.
10984719 - Dna polymerases and sos mutagenesis: can one reconcile the biochemical and genetic data?
20504269 - Quantitative denaturing high performance liquid chromatography (q-dhplc) detection of a...
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-05-14
Journal Detail:
Title:  DNA repair     Volume:  9     ISSN:  1568-7856     ISO Abbreviation:  DNA Repair (Amst.)     Publication Date:  2010 Jul 
Date Detail:
Created Date:  2010-06-28     Completed Date:  2010-09-16     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101139138     Medline TA:  DNA Repair (Amst)     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  835-41     Citation Subset:  IM    
Copyright Information:
2010 Elsevier B.V. All rights reserved.
Affiliation:
Gray Institute for Radiation Oncology & Biology, University of Oxford, United Kingdom.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Casein Kinase II / genetics,  metabolism*
Cell Line
Cytoplasm
DNA Ligases / metabolism
DNA Repair*
DNA-Binding Proteins / metabolism*
Humans
Phosphorylation
Proteasome Endopeptidase Complex
Protein Stability
RNA, Small Interfering / genetics
Ubiquitination
Grant Support
ID/Acronym/Agency:
//Cancer Research UK; //Medical Research Council
Chemical
Reg. No./Substance:
0/DNA-Binding Proteins; 0/RNA, Small Interfering; 0/X-ray repair cross complementing protein 1; EC 2.7.11.1/Casein Kinase II; EC 3.4.25.1/Proteasome Endopeptidase Complex; EC 6.5.1.-/DNA Ligases; EC 6.5.1.-/DNA ligase III

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Model of intestinal chylomicron over-production and Ezetimibe treatment: Impact on the retention of ...
Next Document:  XRCC1 deficiency influences the cytotoxicity and the genomic instability induced by Me-lex, a specif...