Document Detail


X-ray imaging of differential vascular density in MMP-9-/-, PAR-1-/+, hyperhomocysteinemic (CBS-/+) and diabetic (Ins2-/+) mice.
MedLine Citation:
PMID:  20839901     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Although protease activated receptor-1 (PAR-1) and matrix metalloproteinase-9 (MMP-9) play significant role in vascular remodelling in hyperhomocysteinemia (HHcy due to cystathionine beta synthase deficiency, CBS-/+) and diabetes, mechanism remains nebulous. We hypothesized that differential vascular density and remodelling in different vascular beds in HHcy and diabetes were responsible for an adaptive metabolic homeostasis during the pathogenesis. To test this hypothesis, vascular density in mice lacking PAR-1, MMP-9, CBS and Insulin-2 gene mutant (Ins2-/+, Akita) was measured and compared with wild type (WT, C57BL/6J) mice. The vascular density was detected by x-ray angiography using KODAK 4000 MM image station, using barium sulphate as contrasting agent. The % vascular density in the hearts of WT, CBS-/+ (HHcy), MMP-9-/-, PAR-1-/+ and Ins2-/+ (type-1 diabetes) was 100 ± 2.8, 85 ± 3.3, 90 ± 3.3, 95 ± 3.8 and 73 ± 1.7, respectively. The vascular density in CBS-/+ and Akita hearts decreased while it was increased in lungs of CBS-/+ and MMP-9-/-.There was decreased vascular density in liver and kidney of Akita mice. Vascular density in brain, kidney and mesentery was decreased in CBS-/+ mice. These findings support the notation that metabolic derangement in diabetes and HHcy causes the chronic decline and/or rarefaction in vascular density.
Authors:
S Givvimani; U Sen; N Tyagi; C Munjal; S C Tyagi
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2010-09-14
Journal Detail:
Title:  Archives of physiology and biochemistry     Volume:  117     ISSN:  1744-4160     ISO Abbreviation:  Arch. Physiol. Biochem.     Publication Date:  2011 Feb 
Date Detail:
Created Date:  2011-01-06     Completed Date:  2011-04-13     Revised Date:  2011-12-21    
Medline Journal Info:
Nlm Unique ID:  9510153     Medline TA:  Arch Physiol Biochem     Country:  England    
Other Details:
Languages:  eng     Pagination:  1-7     Citation Subset:  IM    
Affiliation:
Department of Physiology and Biophysics, University of Louisville School of Medicine, KY 40202, USA.
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MeSH Terms
Descriptor/Qualifier:
Angiography
Animals
Barium Sulfate / analysis
Blood Vessels* / pathology
Brain / blood supply
Cystathionine beta-Synthase / deficiency,  genetics
Diabetes Mellitus, Type 2* / genetics,  metabolism,  physiopathology
Disease Models, Animal
Hyperhomocysteinemia* / genetics,  metabolism,  physiopathology
Matrix Metalloproteinase 9* / deficiency,  genetics
Mice
Mice, Inbred C57BL
Mice, Transgenic
Organ Size / genetics
Receptor, PAR-1* / deficiency,  genetics
Renal Circulation
Splanchnic Circulation
X-Rays
Grant Support
ID/Acronym/Agency:
HL-71010/HL/NHLBI NIH HHS; HL-74185/HL/NHLBI NIH HHS; HL-88012/HL/NHLBI NIH HHS; NS-51568/NS/NINDS NIH HHS; R01 HL074185-09/HL/NHLBI NIH HHS; R01 HL088012-03/HL/NHLBI NIH HHS; R01 HL108621-02/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
0/Receptor, PAR-1; 7727-43-7/Barium Sulfate; EC 3.4.24.35/Matrix Metalloproteinase 9; EC 4.2.1.22/Cystathionine beta-Synthase

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