Document Detail


Within-woman change in regulatory T cells from pregnancy to the postpartum period.
MedLine Citation:
PMID:  20961621     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Regulatory T cells (Treg cells) are an important area of investigation in human health and disease. In this study, the trajectory of percentage of Treg cells (defined as CD4+CD25+Foxp3+CD127--lymphocytes) was measured in the blood of 208 women during pregnancy and up to three additional times in the postpartum period (1, 6 and 12 months postpartum). Whether the trajectory was affected by gravidity, parity, neonatal sex, pet exposure, maternal atopic and asthma status, smoking, maternal race or other pregnancy factors was examined. Multilevel models were fit using full maximum likelihood methods and included both random and fixed effects. Overall, percentages of Treg cells increased from the prenatal to the postpartum period. Among women who were not atopic, nulliparous women had lower percentages of Treg cells over time compared with parous women. Atopic women with pets in the home during pregnancy had lower percentages of Treg cells than atopic women who did not have pets. The trajectory was not affected by the other factors investigated. We conclude that within-woman change in percentages of Treg cells may vary by time in relation to delivery, as well as by maternal atopic status and exposure to pets and number of prior births. The data did not indicate an overall decline in Treg cells in the postpartum period. Future work to better identify the role of Treg cells in successful pregnancy would ideally include a set of well characterized women sampled serially starting prior to pregnancy and throughout the postpartum period.
Authors:
Ganesa Wegienka; Suzanne Havstad; Kevin R Bobbitt; Kimberley J Woodcroft; Edward M Zoratti; Dennis R Ownby; Christine Cole Johnson
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2010-10-18
Journal Detail:
Title:  Journal of reproductive immunology     Volume:  88     ISSN:  1872-7603     ISO Abbreviation:  J. Reprod. Immunol.     Publication Date:  2011 Jan 
Date Detail:
Created Date:  2010-12-21     Completed Date:  2011-06-28     Revised Date:  2012-01-04    
Medline Journal Info:
Nlm Unique ID:  8001906     Medline TA:  J Reprod Immunol     Country:  Ireland    
Other Details:
Languages:  eng     Pagination:  58-65     Citation Subset:  IM    
Copyright Information:
Copyright © 2010 Elsevier Ireland Ltd. All rights reserved.
Affiliation:
Department of Biostatistics and Research Epidemiology, Henry Ford Hospital, Detroit, MI 48202, USA. gwegien1@hfhs.org
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MeSH Terms
Descriptor/Qualifier:
Antigens, CD4 / genetics
Asthma / immunology
Continental Population Groups
Female
Flow Cytometry
Fluorescent Antibody Technique
Forkhead Transcription Factors / genetics
Gravidity / immunology
Humans
Interleukin-2 Receptor alpha Subunit / genetics
Interleukin-7 Receptor alpha Subunit / genetics,  immunology
Lymphocyte Count
Parity / immunology
Pets
Postpartum Period / genetics,  immunology*
Pregnancy / immunology*
Smoking / immunology
T-Lymphocyte Subsets / immunology*
T-Lymphocytes, Regulatory / immunology*
Grant Support
ID/Acronym/Agency:
AI069271/AI/NIAID NIH HHS; AI070606/AI/NIAID NIH HHS; K01 AI070606-01A2/AI/NIAID NIH HHS; K01 AI070606-02/AI/NIAID NIH HHS; K01 AI070606-03/AI/NIAID NIH HHS; K01 AI070606-04/AI/NIAID NIH HHS; R21 AI069271-01A1/AI/NIAID NIH HHS; R21 AI069271-02/AI/NIAID NIH HHS
Chemical
Reg. No./Substance:
0/Antigens, CD4; 0/FOXP3 protein, human; 0/Forkhead Transcription Factors; 0/Interleukin-2 Receptor alpha Subunit; 0/Interleukin-7 Receptor alpha Subunit

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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