Document Detail


Why does meconium cause meconium aspiration syndrome? Current concepts of MAS pathophysiology.
MedLine Citation:
PMID:  19833459     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
One in every 7 pregnancies ends with meconium-stained amniotic fluid and approximately 5% of these infants develop the meconium aspiration syndrome (MAS). MAS is a severe disease of the (mainly) term neonate, characterized by respiratory distress, pulmonary inflammation, persistent pulmonary hypertension and chronic hypoxia. The pathophysiology of MAS is multifactorial and complex. In this article, we discuss the mechanical and chemical effects of meconium on a newborn's airway, meconium-induced inflammation, mediated by proinflammatory cytokines and chemokines, the complement system and the proinflammatory enzyme phospholipase A2. Furthermore, we focus on MAS-related apoptotic cell death, causing severe acute lung injury due to damage and detachment of lung airway and alveolar cells. Finally, risk factors for MAS development to identify those newborns that develop MAS and those who do not are discussed.
Authors:
Y van Ierland; A J de Beaufort
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Early human development     Volume:  85     ISSN:  1872-6232     ISO Abbreviation:  Early Hum. Dev.     Publication Date:  2009 Oct 
Date Detail:
Created Date:  2009-10-26     Completed Date:  2010-02-12     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  7708381     Medline TA:  Early Hum Dev     Country:  Ireland    
Other Details:
Languages:  eng     Pagination:  617-20     Citation Subset:  IM    
Affiliation:
Division of Neonatology, Juliana Children's Hospital, The Hague, The Netherlands. y.vanierland@erasmusmc.nl
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MeSH Terms
Descriptor/Qualifier:
Apoptosis
Chemokines / physiology
Cytokines / physiology
Humans
Infant, Newborn
Lung / embryology,  pathology
Meconium*
Meconium Aspiration Syndrome / etiology*,  pathology
Phospholipases A2 / physiology
Risk Factors
Chemical
Reg. No./Substance:
0/Chemokines; 0/Cytokines; EC 3.1.1.4/Phospholipases A2

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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