Document Detail


What a disorder: proinflammatory signaling pathways induced by Helicobacter pylori.
MedLine Citation:
PMID:  20863705     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Infection of gastric epithelial cells with Helicobacter pylori induces strong proinflammatory responses by activating nuclear transcription factors NF-κB and AP-1. Several reports indicate that multiple bacterial factors and cellular molecules are involved in this signaling. Injected peptidoglycan, CagA or OipA and urease, and at least 16 different signaling cascades have been implicated in H. pylori-induced proinflammatory signaling. Many of these reports are contradictory, thus generating a highly puzzling scenario. Here we discuss the pros and cons of the multiple signaling activities in the induction of proinflammatory responses and associated problems, and give suggestions for finding ways out of this dilemma.
Authors:
Steffen Backert; Michael Naumann
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-09-20
Journal Detail:
Title:  Trends in microbiology     Volume:  18     ISSN:  1878-4380     ISO Abbreviation:  Trends Microbiol.     Publication Date:  2010 Nov 
Date Detail:
Created Date:  2010-10-26     Completed Date:  2011-01-07     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9310916     Medline TA:  Trends Microbiol     Country:  England    
Other Details:
Languages:  eng     Pagination:  479-86     Citation Subset:  IM    
Copyright Information:
Copyright © 2010 Elsevier Ltd. All rights reserved.
Affiliation:
University College Dublin, School of Biomolecular and Biomedical Sciences, Belfield Campus, Dublin 4, Ireland. steffen.backert@ucd.ie
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MeSH Terms
Descriptor/Qualifier:
Animals
Gastric Mucosa / immunology,  metabolism,  microbiology*
Helicobacter Infections / immunology,  microbiology*
Helicobacter pylori / immunology*,  metabolism,  pathogenicity
Humans
NF-kappa B / metabolism
Signal Transduction*
Chemical
Reg. No./Substance:
0/NF-kappa B

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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