| Vocal intensity in falsetto phonation of a countertenor: an analysis by synthesis approach. | |
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MedLine Citation:
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PMID: 11572375 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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An analysis by synthesis paradigm was implemented to model glottal airflow and vocal tract acoustics for the falsetto phonation of a trained countertenor. Changes in vocal intensity were measured as a function of subglottal pressure, open quotient of the time-varying glottal airflow pulse, and formant tuning. The contributions of laryngeal adduction (open quotient of the glottal flow pulse) and of formant tuning to intensity change were derived from modeled data. The findings were: (1) Subglottal pressure accounted for almost 90% of the variation in SPL in falsetto phonation. (2) The open quotient of the glottal flow pulse was remarkably constant in these falsetto phonations, and thus did not affect vocal intensity significantly. (3) Formant tuning occurred in two out of nine possibilities for the vowel /a/. These instances did not support the concept of systematic exploitation of formant tuning. |
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Authors:
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K Tom; I R Titze |
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Publication Detail:
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Type: Journal Article; Research Support, U.S. Gov't, P.H.S. |
Journal Detail:
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Title: The Journal of the Acoustical Society of America Volume: 110 ISSN: 0001-4966 ISO Abbreviation: J. Acoust. Soc. Am. Publication Date: 2001 Sep |
Date Detail:
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Created Date: 2001-09-26 Completed Date: 2001-10-11 Revised Date: 2007-11-14 |
Medline Journal Info:
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Nlm Unique ID: 7503051 Medline TA: J Acoust Soc Am Country: United States |
Other Details:
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Languages: eng Pagination: 1667-76 Citation Subset: IM |
Affiliation:
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Department of Speech Communication, California State University Fullerton, 92831, USA. |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Acoustics Biomechanics Glottis / physiology Humans Larynx / physiology Male Middle Aged Models, Biological Music* Phonation* Pressure Pulmonary Ventilation Voice* / physiology |
| Grant Support | |
ID/Acronym/Agency:
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5 R01 DC 00387-08/DC/NIDCD NIH HHS |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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