Document Detail


Vkappa polymorphisms in NOD mice are spread throughout the entire immunoglobulin kappa locus and are shared by other autoimmune strains.
MedLine Citation:
PMID:  20556377     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The diversity of immunoglobulin (Ig) and T cell receptor (TCR) genes available to form the lymphocyte repertoire has the capacity to produce a broad array of both protective and harmful specificities. In type 1 diabetes (T1D), the presence of antibodies to insulin and other islet antigens predicts disease development in both mice and humans, and demonstrate that immune tolerance is lost early in the disease process. Anti-insulin T cells isolated from T1D-prone non-obese diabetic (NOD) mice use polymorphic TCRalpha chains, suggesting that the available T cell repertoire is altered in these autoimmune mice. To probe whether insulin-binding B cells also possess polymorphic V genes, Ig light chains were isolated and sequenced from NOD mice that harbor an Ig heavy chain transgene. Three insulin-binding Vkappa genes were identified, all of which were polymorphic to the closest germline sequence matches present in the GenBank database. Additional analysis of over 300 light chain sequences from multiple sources, including germline DNA, shows that polymorphisms are spread throughout the entire NOD Igkappa locus, as these polymorphic sequences represent 43 distinct Vkappa genes which belong to 14 Vkappa families. Database searches reveal that a majority of polymorphic Vkappa genes identified in NOD are identical to Vkappa genes isolated from SLE-prone NZBxNZW F1 or MRL strains of mice, suggesting that a shared Igkappa haplotype may be present. Predicted amino acid changes preferentially occur in CDR, and thus could alter antigen recognition by the germline B cell repertoire of autoimmune versus non-autoimmune mouse strains.
Authors:
Rachel A Henry; Peggy L Kendall; Emily J Woodward; Chrys Hulbert; James W Thomas
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2010-06-17
Journal Detail:
Title:  Immunogenetics     Volume:  62     ISSN:  1432-1211     ISO Abbreviation:  Immunogenetics     Publication Date:  2010 Aug 
Date Detail:
Created Date:  2010-07-27     Completed Date:  2010-08-20     Revised Date:  2010-12-03    
Medline Journal Info:
Nlm Unique ID:  0420404     Medline TA:  Immunogenetics     Country:  United States    
Other Details:
Languages:  eng     Pagination:  507-20     Citation Subset:  IM    
Affiliation:
Department of Medicine, Division of Rheumatology, Vanderbilt University, Nashville, TN, USA.
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MeSH Terms
Descriptor/Qualifier:
Amino Acid Substitution
Animals
Autoimmunity / genetics*
B-Lymphocytes / immunology
Base Sequence
DNA Primers / genetics
Diabetes Mellitus, Type 1 / genetics,  immunology
Genes, Immunoglobulin Heavy Chain
Genes, Immunoglobulin Light Chain*
Immunoglobulin Variable Region / genetics
Immunoglobulin kappa-Chains / genetics*
Insulin / immunology
Mice
Mice, Inbred NOD
Mice, Transgenic
Molecular Sequence Data
Polymorphism, Genetic*
Sequence Homology, Nucleic Acid
Species Specificity
T-Lymphocytes / immunology
Grant Support
ID/Acronym/Agency:
5 T32 GM08554/GM/NIGMS NIH HHS; 5 T32 HL069765/HL/NHLBI NIH HHS; AI051448/AI/NIAID NIH HHS; CA68485/CA/NCI NIH HHS; DK058404/DK/NIDDK NIH HHS; DK20593/DK/NIDDK NIH HHS; HL65962/HL/NHLBI NIH HHS; K08 DK070924/DK/NIDDK NIH HHS; P30 CA68485/CA/NCI NIH HHS; R01 AI051448-07/AI/NIAID NIH HHS; R01 AI051448-08/AI/NIAID NIH HHS
Chemical
Reg. No./Substance:
0/DNA Primers; 0/Immunoglobulin Variable Region; 0/Immunoglobulin kappa-Chains; 11061-68-0/Insulin

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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