Document Detail


Vibrio vulnificus-induced death of Jurkat T-cells requires activation of p38 mitogen-activated protein kinase by NADPH oxidase-derived reactive oxygen species.
MedLine Citation:
PMID:  18571150     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Vibrio vulnificus, a pathogenic bacterium causing primary septicemia, exhibited cytotoxicity towards Jurkat cells of T-lymphocytes through intracellular reactive oxygen species (ROS) production. Pretreatment of Jurkat T-cells with diphenyleneiodonium chloride (DPI) abolished V. vulnificus-induced ROS generation and bacterial ability to cause cell death. Jurkat T-cells expressing dominant-negative protein of Rac subunit of NADPH oxidase (NOX) did not show increased ROS production and cell death by V. vulnificus. Vibrio vulnificus also triggered phosphorylation of mitogen-activated protein kinases (MAPKs) including p38 and ERK1/2 in Jurkat T-cells. Experiments using inhibitors or small interfering RNAs for each MAPK showed that both MAPKs are involved in V. vulnificus-induced cell death. DPI only blocked the phosphorylation of p38 MAPK in Jurkat T-cells exposed by V. vulnificus. This study demonstrates that V. vulnificus induces death of Jurkat T-cells via ROS-dependent activation of p38 MAPK, and that NOX plays a major role in ROS generation in V. vulnificus-exposed cells.
Authors:
Woo Hyang Kim; Sung Young Goo; Myeong Heon Shin; Se-Jin Chun; Heuiran Lee; Kyu-Ho Lee; Soon-Jung Park
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2008-06-20
Journal Detail:
Title:  Cellular immunology     Volume:  253     ISSN:  1090-2163     ISO Abbreviation:  Cell. Immunol.     Publication Date:    2008 May-Jun
Date Detail:
Created Date:  2008-09-18     Completed Date:  2008-10-06     Revised Date:  2009-11-19    
Medline Journal Info:
Nlm Unique ID:  1246405     Medline TA:  Cell Immunol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  81-91     Citation Subset:  IM    
Affiliation:
Department of Environmental Medical Biology and Institute of Tropical Medicine, The Brain Korea 21 Project, Yonsei University College of Medicine, 134 Shinchon-dong, Seodaemun-gu, Seoul 120-752, Republic of Korea.
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MeSH Terms
Descriptor/Qualifier:
Animals
Cell Death*
Enzyme Activation
Enzyme Inhibitors / metabolism
Humans
Jurkat Cells
Mitogen-Activated Protein Kinase 1 / genetics,  metabolism
Mitogen-Activated Protein Kinase 3 / genetics,  metabolism
NADPH Oxidase / metabolism*
Nitric Oxide / metabolism
Nitric Oxide Synthase Type II / metabolism
Onium Compounds / metabolism
RNA Interference
Reactive Oxygen Species / metabolism*
T-Lymphocytes / enzymology,  microbiology,  physiology*
Vibrio vulnificus / pathogenicity*
p38 Mitogen-Activated Protein Kinases / genetics,  metabolism*
Chemical
Reg. No./Substance:
0/Enzyme Inhibitors; 0/Onium Compounds; 0/Reactive Oxygen Species; 10102-43-9/Nitric Oxide; 244-54-2/diphenyleneiodonium; EC 1.14.13.39/Nitric Oxide Synthase Type II; EC 1.6.3.1/NADPH Oxidase; EC 2.7.11.24/Mitogen-Activated Protein Kinase 1; EC 2.7.11.24/Mitogen-Activated Protein Kinase 3; EC 2.7.11.24/p38 Mitogen-Activated Protein Kinases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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