Document Detail


Ventilatory control in humans: constraints and limitations.
MedLine Citation:
PMID:  17234715     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Below the lactate threshold ((thetaL)), ventilation (V(E))responds in close proportion to CO(2) output to regulate arterial partial pressure of CO(2) (PaCO2). While ventilatory control models have traditionally included proportional feedback (central and carotid chemosensory) and feedforward (central and peripheral neurogenic) elements, the mechanisms involved remain unclear. Regardless, putative control schemes have to accommodate the close dynamic 'coupling' between and V(E) and V(CO2). Above (thetaL), PaCO2 is driven down to constrain the fall of arterial pH by a compensatory hyperventilation, probably of carotid body origin. When V(E) requirements are high (as in highly fit endurance athletes), V(E) can attain limiting proportions. Not only does this impair gas exchange at these work rates, but there may be an associated high metabolic cost for generation of respiratory muscle power, which may be sufficient to divert a fraction of the cardiac output away from the muscles of locomotion to the respiratory muscles, further compromising exercise tolerance.
Authors:
Susan A Ward
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Publication Detail:
Type:  Journal Article; Review     Date:  2007-01-18
Journal Detail:
Title:  Experimental physiology     Volume:  92     ISSN:  0958-0670     ISO Abbreviation:  Exp. Physiol.     Publication Date:  2007 Mar 
Date Detail:
Created Date:  2007-03-15     Completed Date:  2007-05-10     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9002940     Medline TA:  Exp Physiol     Country:  England    
Other Details:
Languages:  eng     Pagination:  357-66     Citation Subset:  IM    
Affiliation:
Institute of Membrane and Systems Biology, University of Leeds, Leeds LS2 9JT, UK. s.a.ward@leeds.ac.uk
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MeSH Terms
Descriptor/Qualifier:
Carbon Dioxide / metabolism
Exercise*
Exercise Tolerance*
Humans
Hyperventilation / metabolism,  physiopathology
Lactic Acid / blood
Models, Biological
Muscle Contraction*
Muscle, Skeletal / metabolism*,  physiopathology
Oxygen Consumption
Pulmonary Gas Exchange
Pulmonary Ventilation*
Respiratory Center / physiopathology
Chemical
Reg. No./Substance:
124-38-9/Carbon Dioxide; 50-21-5/Lactic Acid

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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