Document Detail


Ventilatory compensation for lactacidosis in ponies: role of carotid chemoreceptors and lung afferents.
MedLine Citation:
PMID:  1909315     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
We investigated changes in arterial PCO2 (PaCO2) and pulmonary ventilation (VE) in normal, carotid chemoreceptor-denervated, and hilar nerve-denervated ponies during intravenous lactic acid infusion at rest and treadmill exercise at 1.8 mph-5% grade (mild) and 1.8 mph-15% grade (moderate). Lactic acid, (0.5 M) infusion of 0.10, 0.13, and 0.20 ml.min-1.kg-1 at rest and mild and moderate exercise increased arterial [H+] linearly throughout the 10 min of acid infusion. At 10 min of infusion, arterial [H+] had increased approximately 20 nmol/l (0.2 pH units) for each condition and group. Under most conditions, the temporal pattern of PaCO2 during acid infusion was biphasic. At rest and during mild exercise in all groups, and in carotid chemoreceptor-denervated ponies during moderate exercise, PaCO2 increased approximately 2 Torr (P less than 0.05) during the first 2 min of acid infusion. However, in normal ponies during moderate exercise, PaCO2 was not changed from control in the first 2 min of infusion. Between 2 and 10 min of infusion at rest and mild and moderate exercise in all groups, there was a 5-Torr significant decrease in PaCO2, which did not differ (P greater than 0.10) between groups. VE increased between 15-30 s and 2 min of infusion, but VE changed minimally between 2 and 10 min of infusion at rest and exercise in all groups of ponies. We conclude that lactacidosis does increase VE at rest and submaximal exercise in the pony.(ABSTRACT TRUNCATED AT 250 WORDS)
Authors:
B K Erickson; H V Forster; L G Pan; T F Lowry; D R Brown; M A Forster; A L Forster
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Publication Detail:
Type:  Journal Article; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Journal of applied physiology (Bethesda, Md. : 1985)     Volume:  70     ISSN:  8750-7587     ISO Abbreviation:  J. Appl. Physiol.     Publication Date:  1991 Jun 
Date Detail:
Created Date:  1991-10-08     Completed Date:  1991-10-08     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  8502536     Medline TA:  J Appl Physiol     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  2619-26     Citation Subset:  IM; S    
Affiliation:
Department of Physiology, Medical College of Wisconsin, Milwaukee 53226.
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MeSH Terms
Descriptor/Qualifier:
Acidosis, Lactic / complications,  physiopathology*
Afferent Pathways / physiopathology
Animals
Carbon Dioxide / blood
Carotid Body / physiopathology
Chemoreceptor Cells / physiopathology
Denervation
Horses
Hydrogen-Ion Concentration
Hyperventilation / etiology,  physiopathology
Lung / innervation
Physical Exertion / physiology
Respiration / physiology*
Respiratory Transport / physiology
Grant Support
ID/Acronym/Agency:
5F32HL-07970-02/HL/NHLBI NIH HHS; HL-25739/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
124-38-9/Carbon Dioxide

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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