Document Detail


Unheated Cannabis sativa extracts and its major compound THC-acid have potential immuno-modulating properties not mediated by CB1 and CB2 receptor coupled pathways.
MedLine Citation:
PMID:  16504929     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
There is a great interest in the pharmacological properties of cannabinoid like compounds that are not linked to the adverse effects of Delta(9)-tetrahydrocannabinol (THC), e.g. psychoactive properties. The present paper describes the potential immuno-modulating activity of unheated Cannabis sativa extracts and its main non-psychoactive constituent Delta(9)-tetrahydrocanabinoid acid (THCa). By heating Cannabis extracts, THCa was shown to be converted into THC. Unheated Cannabis extract and THCa were able to inhibit the tumor necrosis factor alpha (TNF-alpha) levels in culture supernatants from U937 macrophages and peripheral blood macrophages after stimulation with LPS in a dose-dependent manner. This inhibition persisted over a longer period of time, whereas after prolonged exposure time THC and heated Cannabis extract tend to induce the TNF-alpha level. Furthermore we demonstrated that THCa and THC show distinct effects on phosphatidylcholine specific phospholipase C (PC-PLC) activity. Unheated Cannabis extract and THCa inhibit the PC-PLC activity in a dose-dependent manner, while THC induced PC-PLC activity at high concentrations. These results suggest that THCa and THC exert their immuno-modulating effects via different metabolic pathways.
Authors:
Kitty C M Verhoeckx; Henrie A A J Korthout; A P van Meeteren-Kreikamp; Karl A Ehlert; Mei Wang; Jan van der Greef; Richard J T Rodenburg; Renger F Witkamp
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Publication Detail:
Type:  Journal Article     Date:  2005-11-07
Journal Detail:
Title:  International immunopharmacology     Volume:  6     ISSN:  1567-5769     ISO Abbreviation:  Int. Immunopharmacol.     Publication Date:  2006 Apr 
Date Detail:
Created Date:  2006-02-28     Completed Date:  2006-04-26     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  100965259     Medline TA:  Int Immunopharmacol     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  656-65     Citation Subset:  IM    
Affiliation:
TNO Pharma, Utrechtseweg 48, PZeist, The Netherlands. Verhoeckx@pharma.tno.nl
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MeSH Terms
Descriptor/Qualifier:
Cannabis / chemistry*,  immunology*
Cell Line
Cells, Cultured
Cyclic AMP / pharmacology
Dose-Response Relationship, Drug
Hot Temperature
Humans
Immunologic Factors*
Macrophage Activation / drug effects
Macrophages / drug effects,  immunology
Plant Extracts / chemistry,  pharmacology
Receptor, Cannabinoid, CB1 / antagonists & inhibitors,  drug effects*
Receptor, Cannabinoid, CB2 / antagonists & inhibitors,  drug effects*
Signal Transduction / drug effects
Tetrahydrocannabinol / pharmacology*
Tumor Necrosis Factor-alpha / metabolism
Type C Phospholipases / metabolism
Chemical
Reg. No./Substance:
0/Immunologic Factors; 0/Plant Extracts; 0/Receptor, Cannabinoid, CB1; 0/Receptor, Cannabinoid, CB2; 0/Tumor Necrosis Factor-alpha; 1972-08-3/Tetrahydrocannabinol; 60-92-4/Cyclic AMP; EC 3.1.4.-/Type C Phospholipases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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