Document Detail


Understanding fragile X syndrome: insights from retarded flies.
MedLine Citation:
PMID:  12086633     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Fragile X syndrome, the most common form of inherited mental retardation, is caused by loss-of-function mutations in the fragile X mental retardation 1 (fmr1) gene. FMR1 is an RNA binding protein that is highly expressed in neurons of the central nervous system. Recent studies in Drosophila indicate that FMR1 plays an important role in synaptogenesis and axonal arborization, which may underlie the observed deficits in flight ability and circadian behavior of fmr1 mutant flies. The relevance of these studies to our understanding of fragile X syndrome is discussed.
Authors:
Fen-Biao Gao
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Neuron     Volume:  34     ISSN:  0896-6273     ISO Abbreviation:  Neuron     Publication Date:  2002 Jun 
Date Detail:
Created Date:  2002-06-27     Completed Date:  2002-07-22     Revised Date:  2005-11-17    
Medline Journal Info:
Nlm Unique ID:  8809320     Medline TA:  Neuron     Country:  United States    
Other Details:
Languages:  eng     Pagination:  859-62     Citation Subset:  IM    
Affiliation:
Gladstone Institute of Neurological Disease and Department of Neurology, University of California, San Francisco, San Francisco, CA 94141, USA. fgao@gladstone.ucsf.edu
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MeSH Terms
Descriptor/Qualifier:
Animals
Drosophila / chemistry,  genetics
Drosophila Proteins / deficiency,  genetics*
Fragile X Mental Retardation Protein
Fragile X Syndrome / genetics*,  metabolism
Mental Retardation / genetics*,  metabolism
Nerve Tissue Proteins / biosynthesis,  genetics
RNA-Binding Proteins*
Sequence Homology
Chemical
Reg. No./Substance:
0/Drosophila Proteins; 0/Nerve Tissue Proteins; 0/RNA-Binding Proteins; 139135-51-6/Fragile X Mental Retardation Protein

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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