Document Detail


Ultrasound-accelerated thrombolysis in arterial and venous peripheral occlusions: fibrinogen level effects.
MedLine Citation:
PMID:  20656221     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
PURPOSE: This study retrospectively assesses whether significantly accelerating thrombolysis with ultrasound affects fibrinogen levels in the treatment of peripheral arterial occlusions. MATERIALS AND METHODS: Between December 2005 and August 2007, 38 limbs in 38 patients (17 women; mean age, 60.5 +/- 19.7 years; age range, 17-94 years) were treated with ultrasound-accelerated thrombolysis for peripheral arterial occlusion (PAO) and deep vein thrombosis (DVT), with serum fibrinogen levels measured at baseline and every 24 hours. All occlusions were treated with alteplase (0.5-1.0 mg/h). RESULTS: Complete or partial lysis was achieved in 92.1% of patients. All patients received thrombolytic therapy with mean infusion time of 42.3 hours (range, 20-96 hours). As part of standard clinical practice, patients were not assessed angiographically overnight. Mean total alteplase dose was 40.6 mg (range, 18-96 mg). Across all patients, the fibrinogen level at the end of infusion decreased by an average of 18.5% from baseline, and no patient exhibited a fibrinogen level < 100 mg/dL during treatment. Fibrinogen depletion was more pronounced among patients with venous occlusions (26.4% from baseline) than those with arterial occlusions (15.8% from baseline). No major hemorrhagic complications occurred. One patient (2.6%) experienced a minor bleeding event at the access site, and use of thrombolytics was discontinued; and one patient with a chronic arterial occlusion and underlying coronary disease who did not respond to thrombolytic therapy, experienced an acute myocardial infarction. Of documented 30-day clinical outcomes in 20 patients, 80.0% remained patent at 30 days. CONCLUSIONS: Ultrasound-accelerated thrombolysis for the treatment of PAO and DVT is associated with a very low complication rate and nominal fibrinogen depletion.
Authors:
Rodney D Raabe
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Journal of vascular and interventional radiology : JVIR     Volume:  21     ISSN:  1535-7732     ISO Abbreviation:  J Vasc Interv Radiol     Publication Date:  2010 Aug 
Date Detail:
Created Date:  2010-07-26     Completed Date:  2010-11-02     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9203369     Medline TA:  J Vasc Interv Radiol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1165-72     Citation Subset:  IM    
Copyright Information:
Copyright (c) 2010 SIR. Published by Elsevier Inc. All rights reserved.
Affiliation:
Department of Radiology, Sacred Heart Medical Center, 101 West Eighth Avenue, PO Box 2555, Spokane, WA 99220-2555, USA. rraabe@inland-imaging.com
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MeSH Terms
Descriptor/Qualifier:
Adolescent
Adult
Aged
Aged, 80 and over
Arterial Occlusive Diseases / blood,  drug therapy,  radiography,  therapy*
Biological Markers / blood
Combined Modality Therapy
Down-Regulation
Drug Administration Schedule
Female
Fibrinogen / metabolism*
Fibrinolytic Agents / administration & dosage*,  adverse effects
Humans
Infusions, Parenteral
Male
Middle Aged
Peripheral Vascular Diseases / blood,  drug therapy,  radiography,  therapy*
Retrospective Studies
Thrombolytic Therapy* / adverse effects
Time Factors
Tissue Plasminogen Activator / administration & dosage*,  adverse effects
Treatment Outcome
Ultrasonic Therapy* / adverse effects
Vascular Patency
Venous Thrombosis / blood,  drug therapy,  radiography,  therapy*
Washington
Young Adult
Chemical
Reg. No./Substance:
0/Biological Markers; 0/Fibrinolytic Agents; 9001-32-5/Fibrinogen; EC 3.4.21.68/Tissue Plasminogen Activator

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