Document Detail


Tuning in to the ‘right’ calcium channel regulation in experimental models of diabetes.
MedLine Citation:
PMID:  20726982     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Elucidation of cellular and molecular mechanisms underlying vascular disease is of fundamental importance to the development of pharmacological agents to target these pathways. Pinho et al. in this issue of the BJP provide highly compelling evidence that the δ isoform of phosphatidyl inositol 3-kinase (PI3K δ) was upregulated and accounted for the increase in L-type, voltage-gated, Ca channel current in aortic vascular smooth muscle (VSM) cells of a mouse model of type 1 diabetes. There are several key issues of broad fundamental significance to this work. Firstly, what is the ‘right’ answer about calcium channel regulation in diabetes? Conflicting reports of increased and decreased Ca channel current may be due to specificity of the vascular bed and species. Then, the time course of diabetic vasculopathy may influence the expression of contractile versus proliferative phenotypes of VSM. Also the metabolic characterization of diabetes may enlighten or confound any study of diabetic vascular disease. These issues need attention to move forward work in this area.
Authors:
M Sturek
Publication Detail:
Type:  Comment; Journal Article    
Journal Detail:
Title:  British journal of pharmacology     Volume:  161     ISSN:  1476-5381     ISO Abbreviation:  Br. J. Pharmacol.     Publication Date:  2010 Dec 
Date Detail:
Created Date:  2010-11-29     Completed Date:  2011-05-18     Revised Date:  2011-12-21    
Medline Journal Info:
Nlm Unique ID:  7502536     Medline TA:  Br J Pharmacol     Country:  England    
Other Details:
Languages:  eng     Pagination:  1455-7     Citation Subset:  IM    
Affiliation:
Department of Cellular and Integrative Physiology, Indiana University School of Medicine, Indianapolis, Indiana 46202-5120, USA. msturek@iupui.edu
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MeSH Terms
Descriptor/Qualifier:
Animals
Atherosclerosis / metabolism
Calcium Channels, L-Type / metabolism*
Clinical Trials as Topic
Diabetes Mellitus, Experimental / metabolism*
Humans
Hyperglycemia / metabolism
Male
Mice
Microvessels / physiopathology
Muscle Contraction
Muscle, Smooth, Vascular / metabolism
Myocytes, Smooth Muscle / metabolism
Vasodilation / physiology
Wasting Syndrome / metabolism
Chemical
Reg. No./Substance:
0/Calcium Channels, L-Type
Comments/Corrections
Comment On:
Br J Pharmacol. 2010 Dec;161(7):1458-71   [PMID:  20942845 ]

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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