Document Detail


Tumor necrosis factor-α and Muc2 mucin play major roles in disease onset and progression in dextran sodium sulphate-induced colitis.
MedLine Citation:
PMID:  21949848     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The sequential events and the inflammatory mediators that characterize disease onset and progression of ulcerative colitis (UC) are not well known. In this study, we evaluated the early pathologic events in the pathogenesis of colonic ulcers in rats treated with dextran sodium sulfate (DSS). Following a lag phase, day 5 of DSS treatment was found clinically most critical as disease activity index (DAI) exhibited an exponential rise with severe weight loss and rectal bleeding. Surprisingly, on days 1-2, colonic TNF-α expression (70-80-fold) and tissue protein (50-fold) were increased, whereas IL-1β only increased on days 7-9 (60-90-fold). Days 3-6 of DSS treatment were characterized by a prominent down regulation in the expression of regulatory cytokines (40-fold for IL-10 and TGFβ) and mucin genes (15-18 fold for Muc2 and Muc3) concomitant with depletion of goblet cell and adherent mucin. Remarkably, treatment with TNF-α neutralizing antibody markedly altered DSS injury with reduced DAI, restoration of the adherent and goblet cell mucin and IL-1β and mucin gene expression. We conclude that early onset colitis is dependent on TNF-α that preceded depletion of adherent and goblet cell mucin prior to epithelial cell damage and these biomarkers can be used as therapeutic targets for UC.
Authors:
Poonam Dharmani; Pearl Leung; Kris Chadee
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2011-09-19
Journal Detail:
Title:  PloS one     Volume:  6     ISSN:  1932-6203     ISO Abbreviation:  PLoS ONE     Publication Date:  2011  
Date Detail:
Created Date:  2011-09-28     Completed Date:  2012-02-03     Revised Date:  2012-05-07    
Medline Journal Info:
Nlm Unique ID:  101285081     Medline TA:  PLoS One     Country:  United States    
Other Details:
Languages:  eng     Pagination:  e25058     Citation Subset:  IM    
Affiliation:
Department of Microbiology, Immunology and Infectious Diseases, Gastrointestinal Research Group, Health Sciences Centre, University of Calgary, Calgary, Alberta, Canada.
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MeSH Terms
Descriptor/Qualifier:
Animals
Blotting, Western
Cells, Cultured
Colitis / chemically induced,  metabolism*,  pathology*
Dextran Sulfate / toxicity*
Disease Progression
Epithelial Cells / metabolism
Goblet Cells / metabolism
Immunoenzyme Techniques
Interleukin-10 / metabolism
Male
Mucin-2 / genetics,  metabolism*
RNA, Messenger / genetics
Rats
Rats, Sprague-Dawley
Real-Time Polymerase Chain Reaction
Tumor Necrosis Factor-alpha / genetics,  metabolism*
Grant Support
ID/Acronym/Agency:
//Canadian Institutes of Health Research
Chemical
Reg. No./Substance:
0/Muc2 protein, mouse; 0/Mucin-2; 0/RNA, Messenger; 0/Tumor Necrosis Factor-alpha; 130068-27-8/Interleukin-10; 9042-14-2/Dextran Sulfate
Comments/Corrections

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