Document Detail


Tuberous sclerosis complex 2 loss-of-function mutation regulates reactive oxygen species production through Rac1 activation.
MedLine Citation:
PMID:  18230340     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The products of the TSC1 (hamartin) and TCS2 (tuberin) tumor suppressor genes negatively regulate cell growth by inhibiting mTOR signaling. Recent research has led to the postulation that tuberin and/or hamartin are involved in tumor migration, presumably through Rho activation. Here we show that LEF-8 cells, which contain a Y1571 missense mutation in tuberin, express higher Rac1 activity than tuberin negative and positive cells. We also provide evidence of obvious lamellipodia formation in LEF-8 cells. Since the production of TSC2(Y1571H) cannot form a hetero-complex with hamartin, we further analyzed another mutant, TSC2(R611Q), which also lacks the ability to form a complex with hamartin. Introducing both forms of mutated TSC2 into COS-1 cells increased Rac1 activity as well as cell motility. We also found these two mutants interacted with Rac1. We further demonstrated that the introduction of mutated TSC2 into COS-1 cells can generate higher reactive oxygen species (ROS). These results indicate that loss-of-function mutated tuberin can activate Rac1 and thereby increase ROS production.
Authors:
Tsukasa Suzuki; Swadesh K Das; Hirohumi Inoue; Machiko Kazami; Okio Hino; Toshiyuki Kobayashi; Raymond S Yeung; Ken-Ichi Kobayashi; Tadahiro Tadokoro; Yuji Yamamoto
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2008-01-28
Journal Detail:
Title:  Biochemical and biophysical research communications     Volume:  368     ISSN:  1090-2104     ISO Abbreviation:  Biochem. Biophys. Res. Commun.     Publication Date:  2008 Mar 
Date Detail:
Created Date:  2008-02-15     Completed Date:  2008-03-10     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0372516     Medline TA:  Biochem Biophys Res Commun     Country:  United States    
Other Details:
Languages:  eng     Pagination:  132-7     Citation Subset:  IM    
Affiliation:
Department of Applied Biology and Chemistry, Tokyo University of Agriculture, 1-1-1 Sakuragaoka, Setagaya-ku, Tokyo 156-8502, Japan.
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MeSH Terms
Descriptor/Qualifier:
Animals
Cell Line
Cell Movement
Cercopithecus aethiops
Enzyme Activation
Mice
Mutation / genetics
Protein Binding
Rats
Reactive Oxygen Species / metabolism*
Tumor Suppressor Proteins / genetics,  metabolism*
rac1 GTP-Binding Protein / genetics,  metabolism*
Chemical
Reg. No./Substance:
0/Reactive Oxygen Species; 0/Tumor Suppressor Proteins; 169027-60-5/tuberous sclerosis complex 2 protein; EC 3.6.5.2/rac1 GTP-Binding Protein

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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