Document Detail


Triptolide inactivates Akt and induces caspase-dependent death in cervical cancer cells via the mitochondrial pathway.
MedLine Citation:
PMID:  20878065     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Triptolide, the main active component of the traditional Chinese herbal medicine Tripterygium wilfordii Hook F, has been shown to have potent immunosuppressive and anti-inflammatory properties. Here, we investigated the pro-apoptotic effect of triptolide in human cervical cancer cells and its underlying mechanisms. Exposure of cervical cancer cells to triptolide induced apoptosis, which was accompanied by loss of mitochondrial membrane potential, caspase processing (caspase-8, -9 and -3), and cleavage of the caspase substrate, poly(ADP-ribose) polymerase. The cytotoxic effects of triptolide were significantly inhibited by the caspase inhibitor, z-VAD-fmk. Triptolide-induced apoptosis was associated with a marked reduction in Akt phosphorylation and was exacerbated by LY294002 (phosphatidylinositol-3'-kinase inhibitor). Conversely, it was attenuated by Akt overexpression. Triptolide-induced apoptosis was also associated with downregulation of Mcl-1 and was significantly inhibited by Mcl-1 overexpression. These findings show that triptolide induces caspase-dependent, mitochondria-mediated apoptosis in cervical cancer cells, in part, by negatively regulating Akt and Mcl-1.
Authors:
Min Jung Kim; Tae Hwa Lee; Sung Han Kim; Yeon-Joo Choi; Jeonghoon Heo; Young-Ho Kim
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  International journal of oncology     Volume:  37     ISSN:  1791-2423     ISO Abbreviation:  Int. J. Oncol.     Publication Date:  2010 Nov 
Date Detail:
Created Date:  2010-09-29     Completed Date:  2011-01-25     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9306042     Medline TA:  Int J Oncol     Country:  Greece    
Other Details:
Languages:  eng     Pagination:  1177-85     Citation Subset:  IM    
Affiliation:
Shinsegae Women's Hospital, Busan, Korea.
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MeSH Terms
Descriptor/Qualifier:
Antineoplastic Agents, Alkylating / pharmacology*
Apoptosis / drug effects*
Blotting, Western
Caspases / drug effects,  metabolism
Cell Line, Tumor
Cell Separation
DNA Fragmentation
Diterpenes / pharmacology*
Epoxy Compounds / pharmacology
Female
Flow Cytometry
Hela Cells
Humans
Membrane Potential, Mitochondrial / drug effects
Mitochondria / drug effects*
Phenanthrenes / pharmacology*
Proto-Oncogene Proteins c-akt / drug effects
Proto-Oncogene Proteins c-bcl-2 / drug effects
Reverse Transcriptase Polymerase Chain Reaction
Transfection
Uterine Cervical Neoplasms / metabolism*
Chemical
Reg. No./Substance:
0/Antineoplastic Agents, Alkylating; 0/Diterpenes; 0/Epoxy Compounds; 0/Phenanthrenes; 0/Proto-Oncogene Proteins c-bcl-2; 0/myeloid cell leukemia sequence 1 protein; 38748-32-2/triptolide; EC 2.7.11.1/Proto-Oncogene Proteins c-akt; EC 3.4.22.-/Caspases

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