Document Detail


Treating inflammation by blocking interleukin-1 in a broad spectrum of diseases.
MedLine Citation:
PMID:  22850787     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Interleukin-1 (IL-1) is a highly active pro-inflammatory cytokine that lowers pain thresholds and damages tissues. Monotherapy blocking IL-1 activity in autoinflammatory syndromes results in a rapid and sustained reduction in disease severity, including reversal of inflammation-mediated loss of sight, hearing and organ function. This approach can therefore be effective in treating common conditions such as post-infarction heart failure, and trials targeting a broad spectrum of new indications are underway. So far, three IL-1-targeted agents have been approved: the IL-1 receptor antagonist anakinra, the soluble decoy receptor rilonacept and the neutralizing monoclonal anti-IL-1β antibody canakinumab. In addition, a monoclonal antibody directed against the IL-1 receptor and a neutralizing anti-IL-1α antibody are in clinical trials.
Authors:
Charles A Dinarello; Anna Simon; Jos W M van der Meer
Related Documents :
22690157 - Biomarker genes for detecting estrogenic activity of endocrine disruptors via estrogen ...
11564427 - Nmda or non-nmda receptor antagonists attenuate increased fos expression in spinal dors...
12204197 - Glial cell plasticity in sensory ganglia induced by nerve damage.
2354357 - 5-hydroxytryptamine immunoreactive varicosities in the lamprey spinal cord have no syna...
9024637 - Novel mechanisms of rtk signal generation.
11319837 - Translocation of the il-1 receptor to focal adhesions is regulated through the c-termin...
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review    
Journal Detail:
Title:  Nature reviews. Drug discovery     Volume:  11     ISSN:  1474-1784     ISO Abbreviation:  Nat Rev Drug Discov     Publication Date:  2012 Aug 
Date Detail:
Created Date:  2012-08-01     Completed Date:  2012-10-10     Revised Date:  2013-05-20    
Medline Journal Info:
Nlm Unique ID:  101124171     Medline TA:  Nat Rev Drug Discov     Country:  England    
Other Details:
Languages:  eng     Pagination:  633-52     Citation Subset:  IM    
Affiliation:
Department of Medicine, University of Colorado Denver, Aurora, Colorado 80045, USA. cdinare333@aol.com
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Anti-Inflammatory Agents / pharmacology*,  therapeutic use
Antibodies, Monoclonal / pharmacology,  therapeutic use
Autoimmune Diseases / drug therapy,  immunology,  physiopathology
Drug Delivery Systems
Drug Design
Humans
Inflammation / drug therapy*,  immunology,  physiopathology
Interleukin 1 Receptor Antagonist Protein / pharmacology,  therapeutic use
Interleukin-1 / antagonists & inhibitors*,  metabolism
Receptors, Interleukin-1 / antagonists & inhibitors*,  metabolism
Recombinant Fusion Proteins / pharmacology,  therapeutic use
Severity of Illness Index
Grant Support
ID/Acronym/Agency:
AI‑15614/AI/NIAID NIH HHS; AR‑45584/AR/NIAMS NIH HHS; CA‑04 6934/CA/NCI NIH HHS; R01 AI015614/AI/NIAID NIH HHS
Chemical
Reg. No./Substance:
0/Anti-Inflammatory Agents; 0/Antibodies, Monoclonal; 0/Interleukin 1 Receptor Antagonist Protein; 0/Interleukin-1; 0/Receptors, Interleukin-1; 0/Recombinant Fusion Proteins; 0/canakinumab; 0/rilonacept
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Revisiting lab-on-a-chip technology for drug discovery.
Next Document:  Strong presence of the high grain protein content allele of NAM-B1 in Fennoscandian wheat.