Document Detail


Transmembrane ion distribution and insulin secretion.
MedLine Citation:
PMID:  2437849     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The sodium dependence of insulin release was studied with the in vitro perfused principal islet of channel catfish. When extracellular sodium was replaced with choline, glucose-induced insulin release and glucose enhancement of arginine-induced insulin release were selectively abolished. Lowering the extracellular pH from 7.6 to 7.1 had a similar selective effect on glucose-induced insulin release. It is suggested that the Na:H antiport is responsible for the sodium dependence of insulin release and that a lowered cytosolic pH preferentially inhibits the glucose sensing mechanism.
Authors:
P Ronner
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Publication Detail:
Type:  In Vitro; Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Annals of the New York Academy of Sciences     Volume:  488     ISSN:  0077-8923     ISO Abbreviation:  Ann. N. Y. Acad. Sci.     Publication Date:  1986  
Date Detail:
Created Date:  1987-06-23     Completed Date:  1987-06-23     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  7506858     Medline TA:  Ann N Y Acad Sci     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  341-55     Citation Subset:  IM    
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MeSH Terms
Descriptor/Qualifier:
Animals
Carrier Proteins / metabolism
Catfishes
Cell Membrane / metabolism
Glucose / pharmacology
Hydrogen-Ion Concentration
Insulin / secretion*
Ion Channels / drug effects,  metabolism*
Islets of Langerhans / drug effects,  physiology*
Sodium / metabolism,  pharmacology
Sodium-Hydrogen Antiporter
Grant Support
ID/Acronym/Agency:
AM-35449/AM/NIADDK NIH HHS; AM-35577/AM/NIADDK NIH HHS
Chemical
Reg. No./Substance:
0/Carrier Proteins; 0/Ion Channels; 0/Sodium-Hydrogen Antiporter; 11061-68-0/Insulin; 50-99-7/Glucose; 7440-23-5/Sodium

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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