Document Detail


Transient catabolic state with reduced IGF-I after antenatal glucocorticoids.
MedLine Citation:
PMID:  17622956     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Glucocorticoid (GC) administration before preterm birth reduces neonatal morbidity but may restrain growth. Here we explored the effect of antenatal GC on nutrient substrates [glucose, FFA, amino acids (AA)], and on IGF-I and IGF-binding protein-1 (IGFBP-1). We analyzed umbilical vein (UV) plasma obtained at birth from 91 preterm newborns that received one course of GC (last exposure 1-1358 h before birth) and 49 newborns that did not. We found that recent GC exposure (-48 h) raised glucose, FFA, and AA concentrations, and the homeostasis model assessment of insulin resistance (HOMA-IR) index, but lowered IGF-I concentrations. The AA surge was greater in newborns with a birth weight z score <0 than in those with a z score >0. Although all AA were transiently increased, the increment was most robust for glutamine and alanine. Shorter duration since GC administration and lower IGF-I concentrations independently predicted AA levels. In conclusion, an antenatal course of GC elicited a transient catabolic state encompassing all nutrient substrates, and a temporary drop in IGF-I concentrations. These changes may explain the growth-inhibitory effects of repeated antenatal GC administration. Future research should clarify the role of IGF-I in the protein-catabolic response to GC.
Authors:
Johan Verhaeghe; Florent Vanstapel; Rita Van Bree; Erik Van Herck; Willy Coopmans
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Pediatric research     Volume:  62     ISSN:  0031-3998     ISO Abbreviation:  Pediatr. Res.     Publication Date:  2007 Sep 
Date Detail:
Created Date:  2007-12-05     Completed Date:  2008-01-25     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0100714     Medline TA:  Pediatr Res     Country:  United States    
Other Details:
Languages:  eng     Pagination:  295-300     Citation Subset:  IM    
Affiliation:
Department of Obstetrics and Gynecology, Health Campus Gasthuisberg, Katholieke Universiteit Leuven, 3000 Leuven, Belgium. johan.verhaeghe@uz.kuleuven.be
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MeSH Terms
Descriptor/Qualifier:
Amino Acids / blood
Birth Weight
Blood Glucose / metabolism
Fatty Acids, Nonesterified / blood
Female
Fetal Blood / chemistry
Fetus / drug effects*,  physiology
Glucocorticoids / pharmacology*
Homeostasis
Humans
Infant, Newborn
Insulin-Like Growth Factor Binding Protein 1 / metabolism*
Insulin-Like Growth Factor I / metabolism*
Male
Pregnancy
Premature Birth*
Chemical
Reg. No./Substance:
0/Amino Acids; 0/Blood Glucose; 0/Fatty Acids, Nonesterified; 0/Glucocorticoids; 0/Insulin-Like Growth Factor Binding Protein 1; 67763-96-6/Insulin-Like Growth Factor I

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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