Document Detail


Transient analysis of cardiopulmonary interactions. II. Systolic events.
MedLine Citation:
PMID:  3378986     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The etiology of the fall in left ventricular stroke volume (LVSV) and arterial pressure with a negative intrathoracic pressure (NITP) during inspiration is controversial. An increase in LV afterload produced by NITP has been proposed as one explanation but is difficult to evaluate if preload is also altered. To test the hypothesis that a systolic event alone, i.e., a change in LV afterload or contractility, can reduce LVSV during inspiration independent of changes in LV preload, a rapid transient NITP confined to systole was produced by electrocardiogram-triggered phrenic nerve stimulation in eight anesthetized dogs. Intrathoracic descending aortic diameters were measured by sonomicrometry to transduce qualitative changes in aortic transmural pressure. With the airway completely obstructed systolic NITP resulted in a decrease in LVSV (-8.1%, P less than 0.001) but an increase in the systolic anteroposterior (0.54 mm, P less than 0.01) and right-to-left (0.45 mm, P less than 0.01) aortic diameters compared with preceding beat. Similar significant changes were observed with the airway unobstructed. These observations are consistent with an increased afterload imposed on the LV reducing LVSV and egress of blood out of the thorax. Prolonging NITP to include both systole and diastole, a profound fall in LVSV is observed, consistent with the independent influences of systolic and diastolic events combining to diminish LVSV.(ABSTRACT TRUNCATED AT 250 WORDS)
Authors:
J Peters; M K Kindred; J L Robotham
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Journal of applied physiology (Bethesda, Md. : 1985)     Volume:  64     ISSN:  8750-7587     ISO Abbreviation:  J. Appl. Physiol.     Publication Date:  1988 Apr 
Date Detail:
Created Date:  1988-07-15     Completed Date:  1988-07-15     Revised Date:  2007-11-15    
Medline Journal Info:
Nlm Unique ID:  8502536     Medline TA:  J Appl Physiol     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  1518-26     Citation Subset:  IM    
Affiliation:
Department of Anesthesiology and Critical Care Medicine, Francis Scott Key Medical Center, Johns Hopkins University Medical Institutions, Baltimore, Maryland 21205.
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MeSH Terms
Descriptor/Qualifier:
Airway Obstruction / physiopathology
Animals
Aorta / physiology
Dogs
Electric Stimulation
Esophagus / physiology
Heart / physiology*
Hemodynamics*
Lung / physiology*
Myocardial Contraction*
Phrenic Nerve / physiology
Systole*

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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