Document Detail


Toll-like receptors at the maternal-fetal interface in normal pregnancy and pregnancy disorders.
MedLine Citation:
PMID:  20367625     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Toll-like receptors (TLR) form the major family of pattern recognition receptors (PRR) that are involved in innate immunity. Innate immune responses against microorganisms at the maternal-fetal interface may have a significant impact on the success of pregnancy, as intrauterine infections have been shown to be strongly associated with certain disorders of pregnancy. At the maternal-fetal interface, TLRs are expressed not only in the immune cells but also in non-immune cells such as trophoblasts and decidual cells; moreover, their expression patterns vary according to the stage of pregnancy. Here, we will describe potential functions of TLRs in these cells, their recognition and response to microorganisms, and their involvement in the innate immunity. The impact of TLR-mediated innate immune response will be discussed via animal model studies, as well as clinical observations.
Authors:
Kaori Koga; Gil Mor
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural; Review     Date:  2010-03-29
Journal Detail:
Title:  American journal of reproductive immunology (New York, N.Y. : 1989)     Volume:  63     ISSN:  1600-0897     ISO Abbreviation:  Am. J. Reprod. Immunol.     Publication Date:  2010 Jun 
Date Detail:
Created Date:  2010-06-22     Completed Date:  2010-10-14     Revised Date:  2011-07-20    
Medline Journal Info:
Nlm Unique ID:  8912860     Medline TA:  Am J Reprod Immunol     Country:  Denmark    
Other Details:
Languages:  eng     Pagination:  587-600     Citation Subset:  IM    
Affiliation:
Department of Obstetrics and Gynecology, University of Tokyo School of Medicine, Tokyo, Japan.
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MeSH Terms
Descriptor/Qualifier:
Animals
Decidua* / immunology,  metabolism
Female
Humans
Maternal-Fetal Exchange / immunology*
Mice
Mice, Inbred BALB C
Obstetric Labor, Premature / etiology,  immunology
Pre-Eclampsia / etiology,  immunology
Pregnancy
Pregnancy Complications* / etiology,  immunology
Toll-Like Receptors / metabolism*
Trophoblasts* / immunology,  metabolism
Grant Support
ID/Acronym/Agency:
3N01 HD23342/HD/NICHD NIH HHS; P01 HD054713-03/HD/NICHD NIH HHS; P01HD054713/HD/NICHD NIH HHS
Chemical
Reg. No./Substance:
0/Toll-Like Receptors
Comments/Corrections

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