Document Detail


To be or NUCB2, is nesfatin the answer?
MedLine Citation:
PMID:  17141625     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Signals from the hypothalamus govern food intake and energy balance. A new study describes nesfatin-1, a hypothalamic and brainstem peptide whose expression decreases during fasting. Although central treatment with nesfatin-1 inhibited food intake and nesfatin-1 blockade increased food intake, the role and mechanism of nesfatin in energy balance remains unclear.
Authors:
Michael A Cowley; Kevin L Grove
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Cell metabolism     Volume:  4     ISSN:  1550-4131     ISO Abbreviation:  Cell Metab.     Publication Date:  2006 Dec 
Date Detail:
Created Date:  2006-12-04     Completed Date:  2007-02-02     Revised Date:  2012-10-25    
Medline Journal Info:
Nlm Unique ID:  101233170     Medline TA:  Cell Metab     Country:  United States    
Other Details:
Languages:  eng     Pagination:  421-2     Citation Subset:  IM    
Affiliation:
Division of Neuroscience, Oregon National Primate Research Center, Oregon Health and Science University, Beaverton, Oregon 97006, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Calcium-Binding Proteins / antagonists & inhibitors,  metabolism*
DNA-Binding Proteins / antagonists & inhibitors,  metabolism*
Eating*
Energy Metabolism* / drug effects
Fasting / metabolism
Gene Expression Regulation / drug effects
Humans
Hypothalamus / metabolism*
Nerve Tissue Proteins / antagonists & inhibitors,  metabolism*,  pharmacology
Rats
Signal Transduction* / drug effects
Chemical
Reg. No./Substance:
0/Calcium-Binding Proteins; 0/DNA-Binding Proteins; 0/Nerve Tissue Proteins; 0/nucleobindin

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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