| Thrombophilia and activated protein C resistance. | |
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MedLine Citation:
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PMID: 9841693 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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In addition to classic vascular insults such as inflammation, trauma, malignancy, and surgery, a number of hereditary coagulation defects predispose patients to a wide array of thrombotic complications. A novel genetic defect in factor V allowing for resistance to its cleavage by activated protein C has recently been implicated in a significant number of cases of familial thrombophilia. A brief case report and review of the literature is presented to familiarize surgeons to this important and quite frequent cause of hypercoagulability. |
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Authors:
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C H Selzman; T A Whitehill; W C Krupski |
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Publication Detail:
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Type: Case Reports; Journal Article; Review |
Journal Detail:
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Title: Annals of vascular surgery Volume: 12 ISSN: 0890-5096 ISO Abbreviation: Ann Vasc Surg Publication Date: 1998 Nov |
Date Detail:
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Created Date: 1998-12-22 Completed Date: 1998-12-22 Revised Date: 2005-11-16 |
Medline Journal Info:
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Nlm Unique ID: 8703941 Medline TA: Ann Vasc Surg Country: UNITED STATES |
Other Details:
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Languages: eng Pagination: 601-4 Citation Subset: IM |
Affiliation:
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Section of Vascular Surgery, University of Colorado Health Sciences Center, Denver, CO, USA. |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Activated Protein C Resistance
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complications*,
genetics Adolescent Humans Male Thrombophilia / etiology* Venous Thrombosis / etiology* |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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