Document Detail


Thrombolysis with recombinant unglycosylated single-chain urokinase-type plasminogen activator (saruplase) in acute myocardial infarction: influence of heparin on early patency rate (LIMITS study). Liquemin in Myocardial Infarction During Thrombolysis With Saruplase.
MedLine Citation:
PMID:  7608436     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
OBJECTIVES: The Liquemin in Myocardial Infarction During Thrombolysis With Saruplase (LIMITS) study was instituted to evaluate and characterize the effect of a prethrombolytic heparin bolus (5,000 IU) on the efficacy and safety of saruplase in patients with acute myocardial infarction. BACKGROUND: Heparin has been used after thrombolytic therapy for acute myocardial infarction to prevent reocclusion of the infarct-related artery. METHODS: The study was designed as a randomized, parallel-group, double-blind, multicenter trial. Patients were treated within 6 h of onset of symptoms with either a bolus of 5,000 IU of heparin (Liquemin) (n = 56, HSH group) or placebo (n = 62, PSH group) before thrombolytic treatment with saruplase given as a 20-mg bolus followed by an infusion of 60 mg over 60 min. Thirty minutes after completion of thrombolysis, an intravenous heparin infusion was administered for 5 days. Before coronary angiography was performed at 6 to 12 h after start of lysis, an additional bolus of 5,000 IU heparin was given to all patients. End points studied were patency of the infarct-related artery, changes in the hemostatic system and bleeding complications. RESULTS: In the HSH group (heparin-saruplase-heparin), 78.6% of patients had an open infarct-related vessel (Thrombolysis in Myocardial Infarction [TIMI] flow grade 2 or 3) compared with 56.5% in the PSH group (placebo-saruplase-heparin) (intention-to-treat analysis, p = 0.01). No significant difference was observed between the two groups with regard to changes in fibrinogen and fibrin/fibrinogen degradation products. A total of eight bleeding complications (14.3%) were observed in the HSH group and five (8.1%) in the PSH group; no cerebrovascular event occurred, and no allergic reaction was reported. A total of 12 patients died during the hospital stay, 3 in the HSH group (5.4%) and 9 in the PSH group (14.5%). CONCLUSIONS: In acute myocardial infarction, the administration of a heparin bolus before thrombolytic therapy with saruplase is associated with a significantly higher patency at angiography 6 to 12 h after the start of thrombolysis without any appreciable increase in risk of bleeding.
Authors:
U Tebbe; J Windeler; I Boesl; H Hoffmann; J Wojcik; M Ashmawy; E R?diger Schwarz; P von Loewis; P Rosemeyer; G Hopkins
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Publication Detail:
Type:  Clinical Trial; Journal Article; Multicenter Study; Randomized Controlled Trial    
Journal Detail:
Title:  Journal of the American College of Cardiology     Volume:  26     ISSN:  0735-1097     ISO Abbreviation:  J. Am. Coll. Cardiol.     Publication Date:  1995 Aug 
Date Detail:
Created Date:  1995-08-17     Completed Date:  1995-08-17     Revised Date:  2010-03-24    
Medline Journal Info:
Nlm Unique ID:  8301365     Medline TA:  J Am Coll Cardiol     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  365-73     Citation Subset:  AIM; IM    
Affiliation:
Gruenenthal GmbH, Aachen, Germany.
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MeSH Terms
Descriptor/Qualifier:
Adult
Aged
Amino Acid Sequence
Confounding Factors (Epidemiology)
Double-Blind Method
Drug Administration Schedule
Drug Therapy, Combination
Enzyme Precursors / therapeutic use*
Female
Fibrinolytic Agents / therapeutic use*
Heparin / administration & dosage,  therapeutic use*
Humans
Injections, Intravenous
Male
Middle Aged
Molecular Sequence Data
Myocardial Infarction / drug therapy*
Recombinant Proteins / therapeutic use
Thrombolytic Therapy*
Time Factors
Treatment Outcome
Urokinase-Type Plasminogen Activator / therapeutic use*
Vascular Patency / drug effects*
Chemical
Reg. No./Substance:
0/Enzyme Precursors; 0/Fibrinolytic Agents; 0/Recombinant Proteins; 9005-49-6/Heparin; 99149-95-8/saruplase; EC 3.4.21.73/Urokinase-Type Plasminogen Activator

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