Document Detail


Testosterone suppresses hepcidin in men: a potential mechanism for testosterone-induced erythrocytosis.
MedLine Citation:
PMID:  20660052     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
CONTEXT: The mechanisms by which testosterone increases hemoglobin and hematocrit are unknown.
OBJECTIVE: The aim was to test the hypothesis that testosterone-induced increase in hematocrit is associated with suppression of the iron regulatory peptide hepcidin.
PARTICIPANTS: Healthy younger men (ages 19-35 yr; n = 53) and older men (ages 59-75 yr; n = 56) were studied.
METHODS: Weekly doses of testosterone enanthate (25, 50, 125, 300, and 600 mg) were administered over 20 wk, whereas endogenous testosterone was suppressed by monthly GnRH agonist administration. Blood and serum parameters from each individual were measured at wk 0, 1, 2, 4, 8, and 20. Longitudinal analyses were performed to examine the relationship between hepcidin, hemoglobin, hematocrit, and testosterone while controlling for potential confounders.
RESULTS: High levels of testosterone markedly suppressed serum hepcidin within 1 wk. Hepcidin suppression in response to testosterone administration was dose-dependent in older men and more pronounced than in young men, and this corresponded to a greater rise in hemoglobin in older men. Serum hepcidin levels at 4 and 8 wk were predictive of change in hematocrit from baseline to peak levels.
CONCLUSION: Testosterone administration is associated with suppression of serum hepcidin. Greater increases in hematocrit in older men during testosterone therapy are related to greater suppression of hepcidin.
Authors:
Eric Bachman; Rui Feng; Thomas Travison; Michelle Li; Gordana Olbina; Vaughn Ostland; Jagadish Ulloor; Anqi Zhang; Shehzad Basaria; Tomas Ganz; Mark Westerman; Shalender Bhasin
Publication Detail:
Type:  Journal Article; Randomized Controlled Trial; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2010-07-21
Journal Detail:
Title:  The Journal of clinical endocrinology and metabolism     Volume:  95     ISSN:  1945-7197     ISO Abbreviation:  J. Clin. Endocrinol. Metab.     Publication Date:  2010 Oct 
Date Detail:
Created Date:  2010-10-07     Completed Date:  2010-11-15     Revised Date:  2012-04-27    
Medline Journal Info:
Nlm Unique ID:  0375362     Medline TA:  J Clin Endocrinol Metab     Country:  United States    
Other Details:
Languages:  eng     Pagination:  4743-7     Citation Subset:  AIM; IM    
Affiliation:
Department of Medicine, Boston University School of Medicine, 670 Albany Street, Boston, Massachusetts 02118, USA. eric.bachman@bmc.org
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MeSH Terms
Descriptor/Qualifier:
Adult
Aged
Aging / blood,  physiology
Antimicrobial Cationic Peptides / blood*,  metabolism
Dose-Response Relationship, Drug
Double-Blind Method
Down-Regulation / drug effects
Drug Administration Schedule
Humans
Injections
Leuprolide / administration & dosage
Male
Middle Aged
Polycythemia / chemically induced*
Testosterone / administration & dosage,  pharmacology*
Young Adult
Grant Support
ID/Acronym/Agency:
P30 AG031679/AG/NIA NIH HHS; U01AG14369/AG/NIA NIH HHS
Chemical
Reg. No./Substance:
0/Antimicrobial Cationic Peptides; 0/hepcidin; 53714-56-0/Leuprolide; 58-22-0/Testosterone
Comments/Corrections

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