Document Detail


Teneurin carboxy (C)-terminal associated peptide-1 inhibits alkalosis-associated necrotic neuronal death by stimulating superoxide dismutase and catalase activity in immortalized mouse hypothalamic cells.
MedLine Citation:
PMID:  17900539     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The teneurins and the teneurin C-terminal-associated peptides (TCAP) are implicated in the regulation of neuron growth and differentiation. However, current observations suggest that TCAP-1 may also have a neuroprotective action during times of pH-induced cellular stress in the brain such as during hypoxia-ischemia and brain alkalosis. To test this hypothesis, we cultured a TCAP-1-responsive mouse hypothalamic cell line, N38, using media buffered at pHs 6.8, 7.4, 8.0 and 8.4 subsequently treated with 100 nM TCAP-1. TCAP-1 significantly inhibited the decline in cell proliferation at pHs 8.0 and 8.4 as determined by direct cell viability assays and decreased the incidence of cells showing necrotic morphology. In addition, TCAP-1 decreased the number of cells undergoing necrosis by 4- to 5-fold as measured by uptake of ethidium homodimer III. Moreover, TCAP-1 significantly decreased the incidence of superoxide radicals and increased superoxide dismutase 1 (SOD1) expression. These results were accompanied by an increase in the SOD copper chaperone expression and increased catalase activity and expression. The results indicate that TCAP may play a neuroprotective role during periods of pH stress by upregulating oxygen radical scavenging systems. Thus, the TCAP-teneurin system may be part of a mechanism to protect neurons during trauma, such as hypoxia and ischemia.
Authors:
Gina Trubiani; Arij Al Chawaf; Denise D Belsham; Dalia Barsyte-Lovejoy; David A Lovejoy
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2007-08-22
Journal Detail:
Title:  Brain research     Volume:  1176     ISSN:  1872-6240     ISO Abbreviation:  Brain Res.     Publication Date:  2007 Oct 
Date Detail:
Created Date:  2007-10-18     Completed Date:  2009-01-16     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0045503     Medline TA:  Brain Res     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  27-36     Citation Subset:  IM    
Affiliation:
Department of Cell and Systems Biology, University of Toronto, Toronto, Ontario, Canada.
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MeSH Terms
Descriptor/Qualifier:
Alkalosis / complications,  enzymology,  physiopathology
Animals
Catalase / metabolism*
Cell Line, Transformed
Cell Survival / drug effects,  physiology
Cytoprotection / drug effects
Free Radical Scavengers / metabolism
Hydrogen-Ion Concentration
Hypothalamus / drug effects,  enzymology*,  physiopathology
Mice
Necrosis / drug therapy*,  prevention & control
Nerve Tissue Proteins / metabolism*,  pharmacology
Neurons / drug effects,  enzymology*
Neuroprotective Agents / metabolism,  pharmacology
Oxidative Stress / drug effects,  physiology
Superoxide Dismutase / metabolism*
Superoxides / metabolism
Up-Regulation / drug effects,  physiology
Chemical
Reg. No./Substance:
0/Free Radical Scavengers; 0/Nerve Tissue Proteins; 0/Neuroprotective Agents; 0/TCAP-1 protein, mouse; 11062-77-4/Superoxides; EC 1.11.1.6/Catalase; EC 1.15.1.-/superoxide dismutase 1; EC 1.15.1.1/Superoxide Dismutase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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