Document Detail


Telomere maintenance in telomerase-positive human ovarian SKOV-3 cells cannot be retarded by complete inhibition of telomerase.
MedLine Citation:
PMID:  12220625     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The two known mechanisms for telomere maintenance in eukaryocytes are telomerase in telomerase-positive cells and alternative lengthening of telomeres (ALT) in telomerase-negative cells. We report here that telomere maintenance in the telomerase-positive human ovarian SKOV-3 cells was not affected by inhibition of telomerase. For comparison, the effect of telomerase inhibitors on telomere maintenance in another telomerase-positive cell line (i.e. human pharynx FaDu cells) and the telomerase-negative human osteosarcoma Saos-2 cells was examined. Telomerase activity was measured using a modified telomeric repeat amplification protocol and telomere length was measured using a solution hybridization-based method and fluorescence in situ hybridization. A reverse transcriptase inhibitor (3'-azido-deoxythymidine or AZT) and an antisense against a component of human telomerase RNA (antisense hTR) were used to inhibit telomerase. FaDu and SKOV-3 cells showed comparable baseline telomerase activity. Telomerase activity in both cells was inhibited about equally by AZT (maximal inhibition of approximately 80%) and by expression of antisense hTR (complete inhibition in SKOV-3 cells and maximal inhibition of approximately 80% in FaDu cells). However, treatment with telomerase inhibitors resulted in approximately 50% telomere shortening in FaDu cells but had no effect on SKOV-3 nor Saos-2 cells. SKOV-3 cells did not show the characteristic features of ALT (i.e. heterogeneous telomere length and promyelocytic leukemia bodies), whereas these ALT features were observed in Saos-2 cells. Collectively, these results suggest the existence of a telomerase-independent mechanism of telomere maintenance in the telomerase-positive SKOV-3 cells.
Authors:
Yuebo Gan; Yiqun Mo; Jeffrey Johnston; Jie Lu; M Guillaume Wientjes; Jessie L-S Au
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Publication Detail:
Type:  Journal Article; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  FEBS letters     Volume:  527     ISSN:  0014-5793     ISO Abbreviation:  FEBS Lett.     Publication Date:  2002 Sep 
Date Detail:
Created Date:  2002-09-10     Completed Date:  2002-11-08     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  0155157     Medline TA:  FEBS Lett     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  10-4     Citation Subset:  IM    
Affiliation:
College of Pharmacy, The Ohio State University, 500 West 12th Avenue, Columbus, OH 43210, USA.
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MeSH Terms
Descriptor/Qualifier:
Cells, Cultured
Enzyme Inhibitors / pharmacology
Female
Humans
Oligonucleotides, Antisense / pharmacology
Osteosarcoma / metabolism,  pathology
Ovary / cytology*,  drug effects,  physiology*
Pharynx / cytology,  physiology
Telomerase / antagonists & inhibitors*,  genetics,  metabolism
Telomere / drug effects,  physiology*
Zidovudine / pharmacology
Grant Support
ID/Acronym/Agency:
R01CA77091/CA/NCI NIH HHS
Chemical
Reg. No./Substance:
0/Enzyme Inhibitors; 0/Oligonucleotides, Antisense; 30516-87-1/Zidovudine; EC 2.7.7.49/Telomerase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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