Document Detail


Telomerase-independent telomere length maintenance in the absence of alternative lengthening of telomeres-associated promyelocytic leukemia bodies.
MedLine Citation:
PMID:  15805271     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Immortal tumor cells and cell lines employ a telomere maintenance mechanism that allows them to escape the normal limits on proliferative potential. In the absence of telomerase, telomere length may be maintained by an alternative lengthening of telomeres (ALT) mechanism. All human ALT cell lines described thus far have nuclear domains of unknown function, termed ALT-associated promyelocytic leukemia bodies (APB), containing promyelocytic leukemia protein, telomeric DNA and telomere binding proteins. Here we describe telomerase-negative human cells with telomeres that contain a substantial proportion of nontelomeric DNA sequences (like telomerase-null Saccharomyces cerevisiae survivor type I cells) and that are maintained in the absence of APBs. In other respects, they resemble typical ALT cell lines: the telomeres are highly heterogeneous in length (ranging from very short to very long) and undergo rapid changes in length. In addition, these cells are capable of copying a targeted DNA tag from one telomere into other telomeres. These data show that APBs are not always essential for ALT-mediated telomere maintenance.
Authors:
Clare L Fasching; Kylie Bower; Roger R Reddel
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Cancer research     Volume:  65     ISSN:  0008-5472     ISO Abbreviation:  Cancer Res.     Publication Date:  2005 Apr 
Date Detail:
Created Date:  2005-04-04     Completed Date:  2005-05-26     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  2984705R     Medline TA:  Cancer Res     Country:  United States    
Other Details:
Languages:  eng     Pagination:  2722-9     Citation Subset:  IM    
Affiliation:
Children's Medical Research Institute, Westmead, Sydney, New South Wales, Australia.
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MeSH Terms
Descriptor/Qualifier:
DNA, Viral / genetics
Humans
Leukemia, Promyelocytic, Acute / enzymology,  genetics*,  pathology
Polymorphism, Restriction Fragment Length
Simian virus 40 / genetics
Telomerase / metabolism*
Telomere / genetics*,  metabolism*
Chemical
Reg. No./Substance:
0/DNA, Viral; EC 2.7.7.49/Telomerase

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