Document Detail


Targeting the recruitment of monocytes and macrophages in renal disease.
MedLine Citation:
PMID:  20620675     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Macrophages convert proinflammatory or anti-inflammatory signals of tissue microenvironments into response mechanisms. These response mechanisms largely derive from evolutionary conserved defense programs of innate host defense, wound healing, and tissue homeostasis. Hence, in many settings these programs lead to renal inflammation and tissue remodeling (ie, glomerulonephritis and sclerosis or interstitial nephritis and fibrosis). There is abundant experimental evidence that blocking macrophage recruitment or macrophage activation can ameliorate renal inflammation and fibrosis. In this review we discuss experimental tools to target renal macrophage recruitment by using antagonists against selectins, chemokines, integrins, or other important cytokines that mediate renal injury via macrophage recruitment, some of these already having been used in clinical trials.
Authors:
Volker Vielhauer; Onkar Kulkarni; Christoph A Reichel; Hans-Joachim Anders
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Review    
Journal Detail:
Title:  Seminars in nephrology     Volume:  30     ISSN:  1558-4488     ISO Abbreviation:  Semin. Nephrol.     Publication Date:  2010 May 
Date Detail:
Created Date:  2010-07-12     Completed Date:  2010-12-02     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8110298     Medline TA:  Semin Nephrol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  318-33     Citation Subset:  IM    
Affiliation:
Klinikum der Universität, Ludwig-Maximilians-University, Munich, Germany.
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MeSH Terms
Descriptor/Qualifier:
Animals
Chemokines / antagonists & inhibitors
Cytokines / antagonists & inhibitors
Dendritic Cells / drug effects,  physiology
Forecasting
Humans
Integrins / antagonists & inhibitors
Kidney Diseases / drug therapy*,  immunology*
Macrophages / drug effects*,  physiology*
Molecular Targeted Therapy*
Monocytes / drug effects*,  physiology*
Selectins
Chemical
Reg. No./Substance:
0/Chemokines; 0/Cytokines; 0/Integrins; 0/Selectins

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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