Document Detail


TGFβ2-mediated production of hyaluronan is important for the induction of epicardial cell differentiation and invasion.
MedLine Citation:
PMID:  20633555     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
In the developing heart, the epicardium is a major source of progenitor cells that contribute to the formation of the coronary vessel system. These epicardial progenitors give rise to the different cellular components of the coronary vasculature by undergoing a number of morphological and physiological changes collectively known as epithelial to mesenchymal transformation (EMT). However, the specific signaling mechanisms that regulate epicardial EMT are yet to be delineated. In this study we investigated the role of TGFβ2 and hyaluronan (HA) during epicardial EMT and how signals from these two molecules are integrated during this important process. Here we show that TGFβ2 induces MEKK3 activation, which in turn promotes ERK1/2 and ERK5 phosphorylation. TGFβ2 also increases Has2 expression and subsequent HA production. Nevertheless, inhibition of MEKK3 kinase activity, silencing of ERK5 or pharmacological disruption of ERK1/2 activation significantly abrogates this response. Thus, TGFβ2 promotes Has2 expression and HA production through a MEKK3/ERK1/2/5-dependent cascade. Furthermore, TGFβ2 is able to induce epicardial cell invasion and differentiation but not proliferation. However, inhibition of MEKK3-dependent pathways, degradation of HA by hyaluronidases or blockade of CD44, significantly impairs the biological response to TGFβ2. Taken together, these findings demonstrate that TGFβ2 activation of MEKK3/ERK1/2/5 signaling modulates Has2 expression and HA production leading to the induction of EMT events. This is an important and novel mechanism showing how TGFβ2 and HA signals are integrated to regulate changes in epicardial cell behavior.
Authors:
Evisabel A Craig; Anita F Austin; Richard R Vaillancourt; Joey V Barnett; Todd D Camenisch
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2010-07-13
Journal Detail:
Title:  Experimental cell research     Volume:  316     ISSN:  1090-2422     ISO Abbreviation:  Exp. Cell Res.     Publication Date:  2010 Dec 
Date Detail:
Created Date:  2010-11-09     Completed Date:  2011-01-26     Revised Date:  2011-11-02    
Medline Journal Info:
Nlm Unique ID:  0373226     Medline TA:  Exp Cell Res     Country:  United States    
Other Details:
Languages:  eng     Pagination:  3397-405     Citation Subset:  IM    
Copyright Information:
Published by Elsevier Inc.
Affiliation:
Department of Pharmacology and Toxicology, The University of Arizona, Tucson, AZ 85721, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Antibodies, Monoclonal / immunology,  pharmacology
Antigens, CD44 / immunology
Cell Line
Cell Movement / drug effects,  physiology*
Epithelial-Mesenchymal Transition / drug effects,  physiology*
Gene Expression / drug effects,  genetics
Glucuronosyltransferase / genetics,  metabolism
Humans
Hyaluronic Acid / metabolism*
Hyaluronoglucosaminidase / pharmacology
MAP Kinase Kinase Kinase 3 / genetics,  metabolism
Mice
Mice, Transgenic
Mitogen-Activated Protein Kinase 1 / antagonists & inhibitors,  metabolism
Mitogen-Activated Protein Kinase 3 / antagonists & inhibitors,  metabolism
Mitogen-Activated Protein Kinase 7 / genetics,  metabolism
Pericardium / cytology*
Phosphorylation / drug effects
RNA, Small Interfering / genetics
Signal Transduction / drug effects,  physiology
Stem Cells / cytology*,  drug effects,  metabolism
Transforming Growth Factor beta2 / pharmacology*
Vimentin / metabolism
Grant Support
ID/Acronym/Agency:
ES04940/ES/NIEHS NIH HHS; HL077493/HL/NHLBI NIH HHS; HL085708/HL/NHLBI NIH HHS; HL095155/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
0/Antibodies, Monoclonal; 0/Antigens, CD44; 0/RNA, Small Interfering; 0/TGFB2 protein, human; 0/Transforming Growth Factor beta2; 0/Vimentin; 9004-61-9/Hyaluronic Acid; EC 2.4.1.-/Has2 protein, mouse; EC 2.4.1.17/Glucuronosyltransferase; EC 2.7.11.24/MAPK1 protein, human; EC 2.7.11.24/Mitogen-Activated Protein Kinase 1; EC 2.7.11.24/Mitogen-Activated Protein Kinase 3; EC 2.7.11.24/Mitogen-Activated Protein Kinase 7; EC 2.7.11.25/MAP Kinase Kinase Kinase 3; EC 2.7.11.25/Map3k3 protein, mouse; EC 3.2.1.35/Hyaluronoglucosaminidase

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