Document Detail


TGFbeta signalling: a complex web in cancer progression.
MedLine Citation:
PMID:  20495575     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The distortion of growth factor signalling is the most important prerequisite in tumour progression. Transforming growth factor-beta (TGFbeta) signalling regulates tumour progression by a tumour cell-autonomous mechanism or through tumour-stroma interaction, and has either a tumour-suppressing or tumour-promoting function depending on cellular context. Such inherent complexity of TGFbeta signalling results in arduous, but promising, assignments for developing therapeutic strategies against malignant tumours. As numerous cellular context-dependent factors tightly maintain the balance of TGFbeta signalling and contribute to the regulation of TGFbeta-induced cell responses, in this Review we discuss how they maintain the balance of TGFbeta signalling and how their collapse leads to tumour progression.
Authors:
Hiroaki Ikushima; Kohei Miyazono
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Review    
Journal Detail:
Title:  Nature reviews. Cancer     Volume:  10     ISSN:  1474-1768     ISO Abbreviation:  Nat. Rev. Cancer     Publication Date:  2010 Jun 
Date Detail:
Created Date:  2010-05-24     Completed Date:  2010-06-03     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101124168     Medline TA:  Nat Rev Cancer     Country:  England    
Other Details:
Languages:  eng     Pagination:  415-24     Citation Subset:  IM    
Affiliation:
Department of Molecular Pathology, Graduate School of Medicine, University of Tokyo, Tokyo 113-0033, Japan.
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MeSH Terms
Descriptor/Qualifier:
Animals
Disease Progression*
Humans
Neoplasm Metastasis
Neoplasms / blood supply,  genetics,  metabolism*,  pathology*
Neovascularization, Pathologic
Signal Transduction*
Transforming Growth Factor beta / genetics,  metabolism*
Chemical
Reg. No./Substance:
0/Transforming Growth Factor beta

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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