Document Detail


TARDBP 3'-UTR variant in autopsy-confirmed frontotemporal lobar degeneration with TDP-43 proteinopathy.
MedLine Citation:
PMID:  19618195     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Pathogenic mutations in the gene encoding TDP-43, TARDBP, have been reported in familial amyotrophic lateral sclerosis (FALS) and, more recently, in families with a heterogeneous clinical phenotype including both ALS and frontotemporal lobar degeneration (FTLD). In our previous study, sequencing analyses identified one variant in the 3'-untranslated region (3'-UTR) of the TARDBP gene in two affected members of one family with bvFTD and ALS and in one unrelated clinically assessed case of FALS. Since that study, brain tissue has become available and provides autopsy confirmation of FTLD-TDP in the proband and ALS in the brother of the bvFTD-ALS family and the neuropathology of those two cases is reported here. The 3'-UTR variant was not found in 982 control subjects (1,964 alleles). To determine the functional significance of this variant, we undertook quantitative gene expression analysis. Allele-specific amplification showed a significant increase of 22% (P < 0.05) in disease-specific allele expression with a twofold increase in total TARDBP mRNA. The segregation of this variant in a family with clinical bvFTD and ALS adds to the spectrum of clinical phenotypes previously associated with TARDBP variants. In summary, TARDBP variants may result in clinically and neuropathologically heterogeneous phenotypes linked by a common molecular pathology called TDP-43 proteinopathy.
Authors:
Michael A Gitcho; Eileen H Bigio; Manjari Mishra; Nancy Johnson; Sandra Weintraub; Marsel Mesulam; Rosa Rademakers; Sumi Chakraverty; Carlos Cruchaga; John C Morris; Alison M Goate; Nigel J Cairns
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2009-07-18
Journal Detail:
Title:  Acta neuropathologica     Volume:  118     ISSN:  1432-0533     ISO Abbreviation:  Acta Neuropathol.     Publication Date:  2009 Nov 
Date Detail:
Created Date:  2009-11-02     Completed Date:  2010-02-25     Revised Date:  2011-09-26    
Medline Journal Info:
Nlm Unique ID:  0412041     Medline TA:  Acta Neuropathol     Country:  Germany    
Other Details:
Languages:  eng     Pagination:  633-45     Citation Subset:  IM    
Affiliation:
Alzheimer's Disease Research Center, Washington University School of Medicine, St. Louis, MO 63110, USA.
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MeSH Terms
Descriptor/Qualifier:
3' Untranslated Regions / genetics*
Aged
Aged, 80 and over
Amyotrophic Lateral Sclerosis / genetics,  metabolism,  pathology
Autopsy / methods
Brain / metabolism,  pathology
Chi-Square Distribution
DNA Mutational Analysis / methods
DNA-Binding Proteins / genetics*,  metabolism*
Female
Frontotemporal Lobar Degeneration* / genetics,  metabolism,  pathology
Genetic Predisposition to Disease*
Genetic Variation / genetics*
Humans
Male
Middle Aged
RNA, Messenger / metabolism
Grant Support
ID/Acronym/Agency:
P01 AG003991-25/AG/NIA NIH HHS; P01-AG03991/AG/NIA NIH HHS; P30 AG013854-09/AG/NIA NIH HHS; P30 AG013854-149003/AG/NIA NIH HHS; P30-AG13854/AG/NIA NIH HHS; P30-NS057105/NS/NINDS NIH HHS; P50 AG005681-26/AG/NIA NIH HHS; P50 AG016574-11/AG/NIA NIH HHS; P50 AG16574/AG/NIA NIH HHS; P50-AG05681/AG/NIA NIH HHS; U01 AG016976-03/AG/NIA NIH HHS; U01-AG16976/AG/NIA NIH HHS
Chemical
Reg. No./Substance:
0/3' Untranslated Regions; 0/DNA-Binding Proteins; 0/RNA, Messenger; 0/protein TDP-43

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