Document Detail


T-cell-receptor-dependent actin regulatory mechanisms.
MedLine Citation:
PMID:  17314246     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Following stimulation, T cells undergo marked changes in actin architecture that are required for productive immune responses. T-cell-receptor-dependent reorganization of the actin cytoskeleton is necessary for the formation of the immunological synapse at the T-cell-antigen-presenting-cell contact site and the distal pole complex at the opposite face of the T cell. Convergence of specific signaling molecules within these two plasma membrane domains facilitates downstream signaling events leading to full T-cell activation. Recent studies have identified many of the relevant actin-regulatory proteins, and significant progress has been made in our understanding of how these proteins choreograph molecular movements associated with T-cell activation. Proteins such as WASp, WAVE2, HS1 and cofilin direct the formation of a cortical actin scaffold at the immune synapse, while actin-binding proteins such as ezrin and moesin direct binding of signaling molecules to actin filaments within the distal pole complex.
Authors:
Yanping Huang; Janis K Burkhardt
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review    
Journal Detail:
Title:  Journal of cell science     Volume:  120     ISSN:  0021-9533     ISO Abbreviation:  J. Cell. Sci.     Publication Date:  2007 Mar 
Date Detail:
Created Date:  2007-02-22     Completed Date:  2007-07-31     Revised Date:  2007-12-03    
Medline Journal Info:
Nlm Unique ID:  0052457     Medline TA:  J Cell Sci     Country:  England    
Other Details:
Languages:  eng     Pagination:  723-30     Citation Subset:  IM    
Affiliation:
Department of Pathology and Laboratory Medicine, Children's Hospital of Philadelphia and University of Pennsylvania School of Medicine, 816D Abramson Research Center, 3615 Civic Center Blvd., Philadelphia, PA 19104, USA.
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MeSH Terms
Descriptor/Qualifier:
Actin Depolymerizing Factors / metabolism,  physiology
Actins / metabolism,  physiology*
Animals
Humans
Lymphocyte Activation / physiology
Microfilament Proteins / metabolism,  physiology
Models, Biological
Receptors, Antigen, T-Cell / physiology*
T-Lymphocytes / metabolism,  physiology
Wiskott-Aldrich Syndrome Protein Family / metabolism,  physiology
Grant Support
ID/Acronym/Agency:
R01-AI 50098/AI/NIAID NIH HHS
Chemical
Reg. No./Substance:
0/Actin Depolymerizing Factors; 0/Actins; 0/Microfilament Proteins; 0/Receptors, Antigen, T-Cell; 0/Wiskott-Aldrich Syndrome Protein Family; 144131-77-1/moesin

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