Document Detail


Synergistic antitumor effect of β-elemene and etoposide is mediated via induction of cell apoptosis and cell cycle arrest in non-small cell lung carcinoma cells.
MedLine Citation:
PMID:  21904777     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
β-Elemene, an anticancer agent, was isolated from the traditional Chinese medicine plant, curcuma aromatica. In this study, we investigated the synergistic antitumor effect of β-elemene and etoposide phosphate (VP-16) in A549 non-small cell lung carcinoma cells. The cells were treated with β-elemene (20 or 50 µg/ml), VP-16 (15 µg/ml) or the combination of both for 24 h. Compared to the treatment with β-elemene or VP-16 alone, an increased antitumor activity was observed with the combination of both, which was mediated by the cleavage of PARP, the up-regulation of Bax, p53 and p21, and the suppression of cyclin D1. These results suggest that the combination of β-elemene and VP-16 may be a promising therapeutic option for lung cancer.
Authors:
Fan Zhang; Ling Xu; Xiujuan Qu; Mingfang Zhao; Bo Jin; Jian Kang; Yunpeng Liu; Xuejun Hu
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2011-07-20
Journal Detail:
Title:  Molecular medicine reports     Volume:  4     ISSN:  1791-3004     ISO Abbreviation:  Mol Med Rep     Publication Date:    2011 Nov-Dec
Date Detail:
Created Date:  2011-09-09     Completed Date:  2012-01-09     Revised Date:  2013-02-22    
Medline Journal Info:
Nlm Unique ID:  101475259     Medline TA:  Mol Med Rep     Country:  Greece    
Other Details:
Languages:  eng     Pagination:  1189-93     Citation Subset:  IM    
Affiliation:
Department of Respiratory Medicine, The First Hospital of China Medical University, Shenyang 110001, PR China.
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MeSH Terms
Descriptor/Qualifier:
Antineoplastic Agents / therapeutic use,  toxicity*
Apoptosis / drug effects*
Carcinoma, Non-Small-Cell Lung / drug therapy*,  metabolism
Cell Cycle Checkpoints / drug effects*
Cell Line, Tumor
Curcuma / chemistry
Cyclin D1 / metabolism
Cyclin-Dependent Kinase Inhibitor p21 / metabolism
Etoposide / analogs & derivatives*,  therapeutic use,  toxicity
Humans
Lung Neoplasms / drug therapy*,  metabolism
Organophosphorus Compounds / therapeutic use,  toxicity*
Poly(ADP-ribose) Polymerases / metabolism
Sesquiterpenes / therapeutic use,  toxicity*
Tumor Suppressor Protein p53 / metabolism
bcl-2-Associated X Protein / metabolism
Chemical
Reg. No./Substance:
0/Antineoplastic Agents; 0/Cyclin-Dependent Kinase Inhibitor p21; 0/Organophosphorus Compounds; 0/Sesquiterpenes; 0/Tumor Suppressor Protein p53; 0/bcl-2-Associated X Protein; 0/beta-elemene; 117091-64-2/etoposide phosphate; 136601-57-5/Cyclin D1; 33419-42-0/Etoposide; EC 2.4.2.30/Poly(ADP-ribose) Polymerases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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