Document Detail


Sustained retention of tetradecylthioacetic acid after local delivery reduces angioplasty-induced coronary stenosis in the minipig.
MedLine Citation:
PMID:  11684079     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
OBJECTIVE: The sulfur containing tetradecylthioacetic acid (TTA) has a profound effect on lipid metabolism and may also exert antioxidant and anti-inflammatory actions and thereby counteract coronary stenosis after angioplasty balloon injury. This study examined the possible modulatory effects of TTA, delivered locally, on coronary stenosis in minipigs and the underlying mechanisms of action. METHODS: Coronary balloon angioplasty injury using an oversized balloon was performed to 40 coronary arteries (20 minipigs, Sus Scrofa, Gammelsroed) followed by delivery of placebo or TTA via a local drug delivery balloon catheter. TTA was radiolabelled in four pigs. Quantitative coronary angiography and intracoronary ultrasound (ICUS) were performed before and after injury, and after 4 weeks of follow-up. The arteries were examined with histomorphometry. The antioxidant and anti-inflammatory effects of TTA were examined on LDL oxidation and stimulated release of interleukin (IL)-2 and IL-10 in human peripheral blood mononuclear cells (PBMC), respectively. RESULTS: Radioactive TTA was present in the coronary wall after 4 weeks. Angiographic minimal luminal diameter (mean+/-S.E.M.) in the placebo and TTA group was 1.3+/-0.1 vs. 2.2+/-0.2 mm (P<0.01) at follow-up, stenosis rate was 55 and 20% (P<0.01). Remodeling was -0.56+/-0.12 in the TTA group and -1.28+/-0.09 in the placebo group (P<0.01). TTA significantly prolonged the lag time of LDL oxidation. In phytohemagglutinin stimulated PBMC, TTA significantly decreased IL-2 levels and increased IL-10 levels suggesting a marked anti-inflammatory net effect. CONCLUSIONS: Local delivery of TTA reduces coronary artery stenosis after PTCA as assessed by both angiographic, histomorphometric and ICUS examinations by influencing vessel remodeling rather than intimal hyperplasia. The underlying mechanism(s) seem to involve antioxidant and anti-inflammatory effects of this fatty acid analogue.
Authors:
R J Pettersen; Z A Muna; K K Kuiper; E Svendsen; F Müller; P Aukrust; R K Berge; J E Nordrehaug
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Cardiovascular research     Volume:  52     ISSN:  0008-6363     ISO Abbreviation:  Cardiovasc. Res.     Publication Date:  2001 Nov 
Date Detail:
Created Date:  2001-10-30     Completed Date:  2002-01-07     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  0077427     Medline TA:  Cardiovasc Res     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  306-13     Citation Subset:  IM    
Affiliation:
Department of Heart Disease, Haukeland University Hospital, N-5021, Bergen, Norway. rpet@haukland.no
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MeSH Terms
Descriptor/Qualifier:
Administration, Topical
Angioplasty, Transluminal, Percutaneous Coronary / adverse effects*
Animals
Anti-Inflammatory Agents / administration & dosage*,  metabolism,  pharmacology
Antioxidants / administration & dosage*,  metabolism,  pharmacology
Cells, Cultured
Copper
Coronary Angiography
Coronary Stenosis / etiology,  metabolism,  prevention & control*
Coronary Vessels / injuries*,  metabolism,  ultrasonography
Female
Humans
Interleukin-10 / analysis
Interleukin-2 / analysis
Leukocytes, Mononuclear / drug effects,  immunology
Lipoproteins, LDL / metabolism
Male
Models, Animal
Oxidation-Reduction
Sulfides / administration & dosage*,  metabolism,  pharmacology
Swine, Miniature
Ultrasonography, Interventional
Chemical
Reg. No./Substance:
0/Anti-Inflammatory Agents; 0/Antioxidants; 0/Interleukin-2; 0/Lipoproteins, LDL; 0/Sulfides; 130068-27-8/Interleukin-10; 2921-20-2/1-(carboxymethylthio)tetradecane; 7440-50-8/Copper

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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