Document Detail


Supplementation with EPA or fish oil for 11 months lowers circulating lipids, but does not delay the onset of diabetes in UC Davis-type 2 diabetes mellitus rats.
MedLine Citation:
PMID:  20735878     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
EPA or fish oil supplementation has been suggested as treatments for the prevention of type 2 diabetes mellitus (T2DM) due to their lipid-lowering and potential insulin-sensitising effects. We investigated the effects of supplementation with EPA (1 g/kg body weight per d) or fish oil (3 g/kg body weight per d) on the age of onset of T2DM and circulating glucose, insulin, lipids, leptin and adiponectin in UC Davis (UCD)-T2DM rats. Animals were divided into three groups starting at 1 month of age: control, EPA and fish oil. All the animals were followed until diabetes onset or for up to 12 months of age. Monthly fasting blood samples were collected for the measurement of glucose, lipids, hormones and C-reactive protein (CRP). Neither EPA nor fish oil delayed the onset of T2DM or altered fasting plasma glucose, insulin, CRP, adiponectin or leptin concentrations. The groups did not differ in energy intake or body weight. Fish oil treatment lowered fasting plasma TAG concentrations by 39 (sd 7) % (P < 0.001) and EPA lowered fasting plasma NEFA concentrations by 23 (sd 5) % (P < 0.05) at 4 months of age compared with the control group. EPA and fish oil lowered fasting plasma cholesterol concentrations at 4 months of age by 19 (sd 4) and 22 (sd 4) % compared with the control group, respectively (both P < 0.01). In conclusion, EPA and fish oil supplementation lowers circulating lipid concentrations, but does not delay the onset of T2DM in UCD-T2DM rats.
Authors:
Bethany P Cummings; Kimber L Stanhope; James L Graham; Steven C Griffen; Peter J Havel
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2010-08-24
Journal Detail:
Title:  The British journal of nutrition     Volume:  104     ISSN:  1475-2662     ISO Abbreviation:  Br. J. Nutr.     Publication Date:  2010 Dec 
Date Detail:
Created Date:  2010-12-01     Completed Date:  2011-01-04     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0372547     Medline TA:  Br J Nutr     Country:  England    
Other Details:
Languages:  eng     Pagination:  1628-34     Citation Subset:  IM    
Affiliation:
Department of Molecular Biosciences, School of Veterinary Medicine, University of California, Davis, CA 95616, USA.
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MeSH Terms
Descriptor/Qualifier:
Age of Onset
Animals
Cholesterol / blood
Diabetes Mellitus, Type 2 / blood*,  prevention & control
Dietary Fats / administration & dosage*
Dietary Supplements*
Eicosapentaenoic Acid / pharmacology*
Fatty Acids, Nonesterified / blood
Fish Oils / pharmacology*
Hypolipidemic Agents / pharmacology*
Lipids / blood*
Male
Rats
Rats, Inbred Strains
Triglycerides / blood
Grant Support
ID/Acronym/Agency:
AT002599/AT/NCCAM NIH HHS; AT002993/AT/NCCAM NIH HHS; AT003645/AT/NCCAM NIH HHS; R01 HL075675/HL/NHLBI NIH HHS; R01 HL091333/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
0/Dietary Fats; 0/Fatty Acids, Nonesterified; 0/Fish Oils; 0/Hypolipidemic Agents; 0/Lipids; 0/Triglycerides; 1553-41-9/Eicosapentaenoic Acid; 57-88-5/Cholesterol

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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