Document Detail


Sulfated polymannuroguluronate, a novel anti-AIDS drug candidate, inhibits HIV-1 Tat-induced angiogenesis in Kaposi's sarcoma cells.
MedLine Citation:
PMID:  17868650     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Kaposi's sarcoma (KS), a neoplasm often associated with iatrogenic and acquired immunosuppression, is characterized by prominent angiogenesis. Angiogenic factors released from KS and host cells and HIV viral products-the protein Tat are reported to be involved in angiogenesis. Mounting evidence further suggests that multiple angiogenic activities of Tat contribute to AIDS-associated Kaposi's sarcoma (AIDS-KS). Herein, we report that sulfated polymannuroguluronate (SPMG), a novel anti-AIDS drug candidate now undergoing phase II clinical trial, significantly eliminated Tat-induced angiogenesis in SLK cells both in vitro and in vivo. SPMG significantly and dose-dependently inhibits proliferation, migration, and tube formation by SLK cells. SPMG also dramatically arrested Tat-driven KDR phosphorylation and blocked the interaction between Tat and integrin beta1, thus inhibiting the phosphorylation of the downstream kinases of FAK, paxillin and MAPKs. In addition, SPMG was noted to block the release of bFGF and VEGF from ECM. All these collectively favor an issue that SPMG functions as a promising therapeutic against Tat-induced angiogenesis and pathologic events relevant to AIDS-KS, which adds novel mechanistic profiling to the anti-AIDS action of SPMG.
Authors:
Cong-Xiao Lu; Jing Li; Yong-Xu Sun; Xin Qi; Qing-Juan Wang; Xian-Liang Xin; Mei-Yu Geng
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2007-06-16
Journal Detail:
Title:  Biochemical pharmacology     Volume:  74     ISSN:  0006-2952     ISO Abbreviation:  Biochem. Pharmacol.     Publication Date:  2007 Nov 
Date Detail:
Created Date:  2007-10-01     Completed Date:  2007-11-19     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0101032     Medline TA:  Biochem Pharmacol     Country:  England    
Other Details:
Languages:  eng     Pagination:  1330-9     Citation Subset:  IM    
Affiliation:
Department of Pharmacy, Yantai Yuhuangding Hospital, Yantai 264000, and Department of Molecular Pharmacology, Marine Drug and Food Institute, School of Medicine and Pharmacy, Ocean University of China, Qingdao, PR China. lucongxiao2005@yahoo.com.cn
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MeSH Terms
Descriptor/Qualifier:
Animals
Anti-HIV Agents / administration & dosage,  pharmacology*,  therapeutic use
Cell Adhesion / drug effects
Cell Line, Tumor
Cell Movement / drug effects
Cell Proliferation / drug effects
Collagen
Disease Models, Animal
Dose-Response Relationship, Drug
Drug Combinations
Escherichia coli / genetics
Fibroblast Growth Factor 2 / metabolism
Gene Products, tat / biosynthesis,  pharmacology*
Glutathione Transferase / metabolism
HIV-1 / metabolism*
Humans
Laminin
Male
Mice
Neovascularization, Pathologic / drug therapy*,  metabolism
Polysaccharides / administration & dosage,  pharmacology*,  therapeutic use
Proteoglycans
Recombinant Fusion Proteins / biosynthesis,  pharmacology*
Sarcoma, Kaposi* / blood supply,  metabolism,  pathology
Vascular Endothelial Growth Factor A / metabolism
Chemical
Reg. No./Substance:
0/Anti-HIV Agents; 0/Drug Combinations; 0/Gene Products, tat; 0/Laminin; 0/Polysaccharides; 0/Proteoglycans; 0/Recombinant Fusion Proteins; 0/VEGFA protein, human; 0/Vascular Endothelial Growth Factor A; 0/sulfated polymannuroguluronate; 103107-01-3/Fibroblast Growth Factor 2; 119978-18-6/matrigel; 9007-34-5/Collagen; EC 2.5.1.18/Glutathione Transferase

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