| Studies on the pathogenesis of cancer hypercalcemia. | |
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MedLine Citation:
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PMID: 898535 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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The pathogenesis of hypercalcemia in cancer continues to challenge the clinical investigator. Some aspects of this subject have been reviewed, notably with respect to the possible roles of prostaglandins and osteoclast activating factor, with particular reference to breast cancer. There is considerable evidence that the former humoral factor is operative and beginning evidence that the latter may be also. The hope in this continued work is that with better understanding of the mechanisms of hypercalcemia and bone loclization of tumors we will be in a far better position to control and interdict this localization. |
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Authors:
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W P Laird Myers; R Bockman |
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Publication Detail:
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Type: Journal Article; Research Support, U.S. Gov't, P.H.S. |
Journal Detail:
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Title: Transactions of the American Clinical and Climatological Association Volume: 88 ISSN: 0065-7778 ISO Abbreviation: Trans. Am. Clin. Climatol. Assoc. Publication Date: 1977 |
Date Detail:
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Created Date: 1977-10-31 Completed Date: 1977-10-31 Revised Date: 2009-11-18 |
Medline Journal Info:
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Nlm Unique ID: 7507559 Medline TA: Trans Am Clin Climatol Assoc Country: UNITED STATES |
Other Details:
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Languages: eng Pagination: 177-90 Citation Subset: IM |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Adult Aged Breast Neoplasms / complications Female Humans Hypercalcemia / complications, etiology*, physiopathology Male Middle Aged Neoplasms / complications* Neoplasms, Experimental / complications Osteoclasts / pathology, physiopathology Parathyroid Hormone / biosynthesis Penile Neoplasms / complications Pleural Neoplasms / complications |
| Chemical | |
Reg. No./Substance:
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0/Parathyroid Hormone |
| Comments/Corrections | |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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