| Stress urinary incontinence: where are we now, where should we go? | |
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MedLine Citation:
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PMID: 8765247 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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Stress urinary incontinence results from specific damage to the muscles, fascial structures, and nerves of the pelvic floor. Scientific data are accumulating about the nature of each of these injuries. As we begin to define the damage occurring in each element of the continence mechanism, we should be able to precisely select treatment plans on the basis of the abnormality found in individual patients. For example, a woman who has lost all neural control of her pelvic muscles could be saved the useless frustration of attempting pelvic muscle strengthening, whereas a woman with intact but weak muscles can be made continent with exercise. Before these advances can be realized, we must change our current empiric approach that assigns women to treatment because they have stress urinary incontinence to one that asks about the status of each part of the continence mechanism. |
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Authors:
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J O DeLancey |
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Publication Detail:
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Type: Journal Article; Research Support, U.S. Gov't, P.H.S.; Review |
Journal Detail:
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Title: American journal of obstetrics and gynecology Volume: 175 ISSN: 0002-9378 ISO Abbreviation: Am. J. Obstet. Gynecol. Publication Date: 1996 Aug |
Date Detail:
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Created Date: 1996-11-08 Completed Date: 1996-11-08 Revised Date: 2007-11-14 |
Medline Journal Info:
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Nlm Unique ID: 0370476 Medline TA: Am J Obstet Gynecol Country: UNITED STATES |
Other Details:
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Languages: eng Pagination: 311-9 Citation Subset: AIM; IM |
Affiliation:
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Department of Obstetrics and Gynecology, University of Michigan Medical School, Ann Arbor, USA. |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Female Gynecology / trends Humans Neuromuscular Junction / physiopathology Urethra / physiopathology Urinary Bladder / physiopathology Urinary Incontinence, Stress / physiopathology*, therapy* Urinary Tract / innervation |
| Grant Support | |
ID/Acronym/Agency:
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P30 AG 08808/AG/NIA NIH HHS; R01 DK 47516/DK/NIDDK NIH HHS; R01 NR 04007/NR/NINR NIH HHS |
| Comments/Corrections | |
Comment In:
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Am J Obstet Gynecol. 1997 Dec;177(6):1561
[PMID:
9423778
]
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From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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