Document Detail


Stress-induced apoptosis is impaired in cells with a lysosomal targeting defect but is not affected in cells synthesizing a catalytically inactive cathepsin D.
MedLine Citation:
PMID:  12934083     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The role of cathepsin D in stress-induced cell death has been investigated by using ovine fibroblasts exhibiting a missense mutation in the active site of cathepsin D. The cathepsin D (lysosomal aspartic protease) deficiency did not protect cells against toxicity induced by doxorubicin and other cytotoxic agents, neither did it protect cells from caspase activation. Moreover, the cathepsin D inhibitor, pepstatin A, did not prevent stress-induced cell death in human fibroblasts or lymphoblasts. The possible role of lysosomal ceramide or sphingosine-mediated activation of cathepsin D in apoptosis was also excluded by using human cells either overexpressing or deficient in acid ceramidase. However, a normal lysosomal function seems to be required for efficient cell death, as indicated by the finding that fibroblasts from patients with mucolipidosis II were partially resistant to staurosporine, sphingosine and TNF-induced apoptosis, suggesting a key role of lysosomes in cell death.
Authors:
C Tardy; J Tyynelä; A Hasilik; T Levade; N Andrieu-Abadie
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Cell death and differentiation     Volume:  10     ISSN:  1350-9047     ISO Abbreviation:  Cell Death Differ.     Publication Date:  2003 Sep 
Date Detail:
Created Date:  2003-08-22     Completed Date:  2004-04-16     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  9437445     Medline TA:  Cell Death Differ     Country:  England    
Other Details:
Languages:  eng     Pagination:  1090-100     Citation Subset:  IM    
Affiliation:
INSERM U466, Institut Louis Bugnard, Centre Hospitalier Universitaire de Rangueil, Toulouse, France.
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MeSH Terms
Descriptor/Qualifier:
Acid Ceramidase
Amidohydrolases / genetics
Animals
Apoptosis*
Caspases / metabolism
Catalytic Domain
Cathepsin D / biosynthesis,  genetics,  physiology*
Cathepsins / antagonists & inhibitors
Cells, Cultured
Ceramidases
Ceramides / metabolism
Doxorubicin / toxicity
Fibroblasts / cytology,  drug effects,  metabolism
Humans
Lysosomes / enzymology*
Mucolipidoses / pathology
Mutation, Missense
Pepstatins / pharmacology
Protease Inhibitors / pharmacology
Protein Transport
Sheep
Chemical
Reg. No./Substance:
0/Ceramides; 0/Pepstatins; 0/Protease Inhibitors; 23214-92-8/Doxorubicin; 39324-30-6/pepstatin; EC 3.4.-/Cathepsins; EC 3.4.22.-/Caspases; EC 3.4.23.5/Cathepsin D; EC 3.5.-/Amidohydrolases; EC 3.5.1.23/ASAH1 protein, human; EC 3.5.1.23/Acid Ceramidase; EC 3.5.1.23/Ceramidases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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