Document Detail


Stress in early pregnancy: maternal neuro-endocrine-immune responses and effects.
MedLine Citation:
PMID:  20079933     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Stress profoundly compromises reproduction, particularly when experienced in early gestation. One outcome is pregnancy failure: although glucocorticoids have adverse effects it is not clear what their role in pregnancy failure is. However, secretion of vital hormones such as progesterone and prolactin are reduced and this unbalances the delicate and important pregnancy-protective cytokine milieu. Complex interaction between glucocorticoids, progesterone/prolactin and the immune system evidently precipitate the loss, although early loss may confer reproductive advantage by preserving maternal energy stores and facilitating ongoing maternal care for other offspring. If pregnancy failure is not induced another, perhaps more profound, outcome of maternal stress is fetal programming. Much is known about the role of elevated glucocorticoids during late gestation in fetal programming, but in early gestation their role is less clear, though likely. Other key pregnancy hormones and immune factors also contribute to fetal programming. Undoubtedly integrated action of glucocorticoids, progesterone/prolactin and the immune system is crucial for optimal pregnancy outcome and is highly susceptible to environmental conditions.
Authors:
Victoria J Parker; Alison J Douglas
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Review     Date:  2010-01-15
Journal Detail:
Title:  Journal of reproductive immunology     Volume:  85     ISSN:  1872-7603     ISO Abbreviation:  J. Reprod. Immunol.     Publication Date:  2010 May 
Date Detail:
Created Date:  2010-04-23     Completed Date:  2010-07-21     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8001906     Medline TA:  J Reprod Immunol     Country:  Ireland    
Other Details:
Languages:  eng     Pagination:  86-92     Citation Subset:  IM    
Copyright Information:
Copyright (c) 2009 Elsevier Ireland Ltd. All rights reserved.
Affiliation:
Centre for Integrative Physiology, University of Edinburgh, Edinburgh, UK.
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MeSH Terms
Descriptor/Qualifier:
Animals
Female
Humans
Hypothalamo-Hypophyseal System / immunology*
Neuroimmunomodulation / immunology*
Neurosecretory Systems / immunology*
Pituitary-Adrenal System / immunology*
Pregnancy / immunology*
Stress, Physiological / immunology*
Grant Support
ID/Acronym/Agency:
//Biotechnology and Biological Sciences Research Council

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