Document Detail

Stationary domain wall contribution to enhanced ferroelectric susceptibility.
MedLine Citation:
PMID:  24430571     Owner:  NLM     Status:  In-Data-Review    
In ferroelectrics, the effect of domain wall motion on properties has been widely studied, but non-motional or stationary contributions from the volume of material within the domain wall itself has received less attention. Here we report the measurement of stationary domain wall contributions to permittivity in PbZr0.2Ti0.8O3 films. Studies of (001)-, (101)- and (111)-oriented epitaxial films reveal that (111)-oriented films, in which the motional domain wall contributions are frozen out, exhibit permittivity values approximately three times larger than the intrinsic response alone. This discrepancy can only be accounted for by considering a stationary contribution from the domain wall volume of the material that is 6-78 times larger than the bulk response, and is consistent with predictions of the enhancement of susceptibilities within 90° domain walls. This work offers new insights into the microscopic origin of dielectric enhancement and provides a pathway to engineer the dielectric response of materials.
Ruijuan Xu; J Karthik; Anoop R Damodaran; Lane W Martin
Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Nature communications     Volume:  5     ISSN:  2041-1723     ISO Abbreviation:  Nat Commun     Publication Date:  2014 Jan 
Date Detail:
Created Date:  2014-01-16     Completed Date:  -     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101528555     Medline TA:  Nat Commun     Country:  England    
Other Details:
Languages:  eng     Pagination:  3120     Citation Subset:  IM    
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine

Previous Document:  Artificial inhibitory effects of sulfite on photosystem II activity measured by oxygen evolution in ...
Next Document:  Mitochondrial haplotypes may modulate the phenotypic manifestation of the LHON-associated ND1 G3460A...