Document Detail


Stability of substantia nigra pars reticulata neuronal discharge rates during dopamine receptor blockade and its possible mechanisms.
MedLine Citation:
PMID:  16791106     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
It is hypothesized that substantia nigra pars reticulata neurons become overactive during a deficit of dopamine transmission. In this study, we examined how acute dopamine receptor blockade (SCH23390 and eticlopride) affects impulse activity of substantia nigra pars reticulata neurons and their response to iontophoretic gamma-amino-n-butyric acid in awake, unrestrained rats. No changes in discharge rate were found during complete dopamine receptor blockade, but these neurons showed a diminished response to gamma-amino-n-butyric acid, suggesting gamma-amino-n-butyric acid receptor hyposensitivity. This may result from tonic increase in gamma-amino-n-butyric acid input from the striatum and globus pallidus, which are activated during dopamine receptor blockade. As substantia nigra pars reticulata neurons are autoactive and resistant to tonic increases in gamma-amino-n-butyric acid input, changes in their responsiveness to phasic gamma-amino-n-butyric acid inputs, not tonic increase discharge rate, may underlie movement disturbance following dopamine deficit.
Authors:
François Windels; Eugene A Kiyatkin
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Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, N.I.H., Intramural    
Journal Detail:
Title:  Neuroreport     Volume:  17     ISSN:  0959-4965     ISO Abbreviation:  Neuroreport     Publication Date:  2006 Jul 
Date Detail:
Created Date:  2006-06-22     Completed Date:  2006-09-08     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  9100935     Medline TA:  Neuroreport     Country:  England    
Other Details:
Languages:  eng     Pagination:  1071-5     Citation Subset:  IM    
Affiliation:
Cellular Neurobiology Branch, National Institute on Drug Abuse - Intramural Research Program, National Institutes of Health, DHHS, Baltimore, Maryland 21224, USA.
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MeSH Terms
Descriptor/Qualifier:
Action Potentials / drug effects,  physiology*
Animals
Benzazepines / pharmacology
Dopamine Antagonists / pharmacology
Dose-Response Relationship, Drug
Drug Interactions
Male
Neurons / drug effects,  physiology*
Rats
Rats, Long-Evans
Receptors, Dopamine / physiology*
Salicylamides / pharmacology
Substantia Nigra / cytology*
gamma-Aminobutyric Acid / pharmacology
Chemical
Reg. No./Substance:
0/Benzazepines; 0/Dopamine Antagonists; 0/Receptors, Dopamine; 0/Salicylamides; 56-12-2/gamma-Aminobutyric Acid; 84226-12-0/eticlopride

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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