Document Detail


Somatostatin antagonism prevents elemental diet-induced intestinal atrophy in the rat.
MedLine Citation:
PMID:  11575454     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Somatostatin is a peptide with known antiproliferative effects on the intestine. The aim of the present work was to determine whether somatostatin (SST) antagonism reduces elemental diet-induced intestinal atrophy in the rat. Male Wistar rats were fed a standard diet and treated for seven days with either continuous infusion of saline or low and high doses of a putative somatostatin antagonist; another group was given a SST antagonist in a pulsatile high dose. All these groups received an elemental diet to induce gut mucosa atrophy. Rats were killed and samples were obtained for morphometric and proliferative measurements of the intestine and for SST and insulin-like growth factor-1 (IGF-1) level determination. The elemental diet decreased mucosal length and proliferation. Pulsatile administration of SST antagonist improved or prevented both effects, whereas continuous SST antagonist delivery prevented decreased crypt proliferation induced by the elemental diet. Somatostatin plasma levels were lowest in rats receiving pulsatile administration of SST antagonist. In conclusion, somatostatin antagonism increases proliferation in the intestinal mucosa, improving elemental diet-induced intestinal atrophy; however, morphological growth is not affected.
Authors:
I A Gómez de Segura; T Castell; I Vázquez; A Mata; E De Miguel
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Digestive diseases and sciences     Volume:  46     ISSN:  0163-2116     ISO Abbreviation:  Dig. Dis. Sci.     Publication Date:  2001 Sep 
Date Detail:
Created Date:  2001-09-28     Completed Date:  2001-10-18     Revised Date:  2003-11-14    
Medline Journal Info:
Nlm Unique ID:  7902782     Medline TA:  Dig Dis Sci     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1985-92     Citation Subset:  AIM; IM    
Affiliation:
Department of Experimental Surgery, La Paz University Hospital, Madrid, Spain.
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MeSH Terms
Descriptor/Qualifier:
Adaptation, Physiological
Animals
Atrophy
Cell Division
Food, Formulated
Insulin-Like Growth Factor I / analysis,  physiology
Intestinal Mucosa / pathology*,  physiopathology
Male
Rats
Rats, Wistar
Receptors, Somatostatin / antagonists & inhibitors*
Somatostatin / physiology*
Chemical
Reg. No./Substance:
0/Receptors, Somatostatin; 51110-01-1/Somatostatin; 67763-96-6/Insulin-Like Growth Factor I

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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