Document Detail


Smoothelin-like 1 protein is a bifunctional regulator of the progesterone receptor during pregnancy.
MedLine Citation:
PMID:  21771785     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
During pregnancy, uterine smooth muscle (USM) coordinately adapts its contractile phenotype in order to accommodate the developing fetus and then prepare for delivery. Herein we show that SMTNL1 plays a major role in pregnancy to promote adaptive responses in USM and that this process is specifically mediated through interactions of SMTNL1 with the steroid hormone receptor PR-B. In vitro and in vivo SMTNL1 selectively binds PR and not other steroid hormone receptors. The physiological relationship between the two proteins was also established in global gene expression and transcriptional reporter studies in pregnant smtnl1(-/-) mice and by RNA interference in progesterone-sensitive cell lines. We show that the contraction-associated and progestin-sensitive genes (oxytocin receptor, connexin 43, and cyclooxygenase-2) and prolactins are down-regulated in pregnant smtnl1(-/-) mice. We suggest that SMTNL1 is a bifunctional co-regulator of PR-B signaling and thus provides a molecular mechanism whereby PR-B is targeted to alter gene expression patterns within USM cells to coordinately promote alterations in USM function during pregnancy.
Authors:
Khaldon Bodoor; Beata Lontay; Rachid Safi; Douglas H Weitzel; David Loiselle; Zhengzheng Wei; Szabolcs Lengyel; Donald P McDonnell; Timothy A Haystead
Related Documents :
15516385 - Trends in congenital malformations, 1974-1999: effect of prenatal diagnosis and electiv...
17559185 - Transvaginal sonographic findings of a large intramural uterine hematoma associated wit...
12660265 - Changes in circulating concentrations of inhibins a and pro-alpha c during first trimes...
17154635 - Dependence of patterned binary alkanethiolate self-assembled monolayers on "uv-photopat...
6512785 - Maternal addison's disease and fetal growth retardation. a case report.
8616965 - The diagnosis of preterm labor and the prediction of preterm delivery.
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2011-07-19
Journal Detail:
Title:  The Journal of biological chemistry     Volume:  286     ISSN:  1083-351X     ISO Abbreviation:  J. Biol. Chem.     Publication Date:  2011 Sep 
Date Detail:
Created Date:  2011-09-05     Completed Date:  2011-12-05     Revised Date:  2011-12-09    
Medline Journal Info:
Nlm Unique ID:  2985121R     Medline TA:  J Biol Chem     Country:  United States    
Other Details:
Languages:  eng     Pagination:  31839-51     Citation Subset:  IM    
Affiliation:
Department of Pharmacology and Cancer Biology, Duke University Medical Center, Durham, North Carolina 27710, USA.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Female
Gene Expression Profiling
Gene Expression Regulation / physiology
Mice
Muscle Contraction / physiology
Muscle Proteins / metabolism,  physiology*
Muscle, Smooth / metabolism
Myometrium / metabolism,  physiology
Phosphoproteins / metabolism,  physiology*
Pregnancy
Progestins
Prolactin
Receptors, Progesterone / metabolism*
Transcription, Genetic
Grant Support
ID/Acronym/Agency:
R37 DK048807-18/DK/NIDDK NIH HHS; R56DK065954-05/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/CHASM protein, mouse; 0/Muscle Proteins; 0/Phosphoproteins; 0/Progestins; 0/Receptors, Progesterone; 9002-62-4/Prolactin

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  SLC6A14 (ATB0,+), a highly concentrative and broad-specific amino acid transporter, is a novel and e...
Next Document:  Resistance to inhibitors of cholinesterase-8A (Ric-8A) is critical for growth factor receptor-induce...