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Sitagliptin Promotes Macrophage-to-feces Reverse Cholesterol Transport Through Reduced Intestinal Cholesterol Absorption in Obese Insulin Resistant CETP-apoB100 Transgenic Mice.
MedLine Citation:
PMID:  22268579     Owner:  NLM     Status:  Publisher    
Abstract/OtherAbstract:
Dipeptidyl peptidase-4 (DPP-4) inhibitors improve glycemic control in type 2 diabetes, but their benefits on reverse cholesterol transport (RCT) remain unknown. We evaluated the effects of DPP-4 inhibitor sitagliptin 500mg/kg/day on RCT in obese insulin resistant CETP-apoB100 transgenic mice. Metformin 300mg/kg/day orally was used as a reference compound. Both metformin and sitagliptin showed the expected effects on glucose parameters. Although no significant effect was observed on total cholesterol and HDL-c levels, sitagliptin, but not metformin, increased fecal cholesterol mass excretion by 132% (p<0.001 vs. vehicle), suggesting a potent effect on cholesterol metabolism. Mice were then injected i.p. with (3) H-cholesterol labelled macrophages to measure RCT over 48 hours. Compared to vehicle, sitagliptin significantly increased macrophage-derived (3) H-cholesterol fecal excretion by 39%. Administration of (14) C-cholesterol labelled olive oil orally showed a significant reduction of (14) C-tracer plasma appearance over time with sitagliptin, indicating that this drug promotes RCT through reduced intestinal cholesterol absorption.
Authors:
François Briand; Quentin Thieblemont; Rémy Burcelin; Thierry Sulpice
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Publication Detail:
Type:  JOURNAL ARTICLE     Date:  2012-1-23
Journal Detail:
Title:  Diabetes, obesity & metabolism     Volume:  -     ISSN:  1463-1326     ISO Abbreviation:  -     Publication Date:  2012 Jan 
Date Detail:
Created Date:  2012-1-24     Completed Date:  -     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  100883645     Medline TA:  Diabetes Obes Metab     Country:  -    
Other Details:
Languages:  ENG     Pagination:  -     Citation Subset:  -    
Copyright Information:
© 2012 Blackwell Publishing Ltd.
Affiliation:
Physiogenex SAS, Prologue Biotech, Rue Pierre et Marie Curie, B.P. 28262, 31682 Labège-Innopole, France. INSERM U1048, I2MC, 1 avenue Jean Poulhès, B.P. 84225, 31400 Toulouse, France.
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