Document Detail


Silencing of MGMT expression by promoter hypermethylation in the metaplasia-dysplasia-carcinoma sequence of Barrett's esophagus.
MedLine Citation:
PMID:  19027227     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
To determine the relevance of MGMT in Barrett's carcinogenesis, we analyzed promotor hypermethylation and expression of MGMT in Barrett's adenocarcinomas and its paired precursor lesions from 133 patients using a methylation-specific PCR, real-time RT-PCR and immunohistochemistry. Hypermethylation was detected in 78.9% of esophageal adenocarcinomas, in 100% of Barrett's intraepithelial neoplasia, in 88.9% of Barrett's metaplasia, but only in 21.4% of normal esophageal mucosa samples (P<0.001) and correlated significantly with downregulation of MGMT transcripts (P=0.048) and protein expression (P=0.02). Decrease of protein expression was significantly correlated with progressed stage of disease, lymph node invasion and tumor size. We conclude, that aberrant promoter methylation of MGMT is a frequent and early event during tumorigenesis of Barrett's esophagus. High prevalence of MGMT hypermethylation may represent a candidate marker for improved diagnosis and targeted therapy in Barrett's adenocarcinoma.
Authors:
Doerthe Kuester; Wa'el El-Rifai; DunFa Peng; Petra Ruemmele; Ivonne Kroeckel; Brigitte Peters; Christopher A Moskaluk; Manfred Stolte; Klaus Mönkemüller; Frank Meyer; Hans-Ulrich Schulz; Arndt Hartmann; Albert Roessner; Regine Schneider-Stock
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2008-11-21
Journal Detail:
Title:  Cancer letters     Volume:  275     ISSN:  1872-7980     ISO Abbreviation:  Cancer Lett.     Publication Date:  2009 Mar 
Date Detail:
Created Date:  2009-01-28     Completed Date:  2009-02-12     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  7600053     Medline TA:  Cancer Lett     Country:  Ireland    
Other Details:
Languages:  eng     Pagination:  117-26     Citation Subset:  IM    
Affiliation:
Department of Pathology, Otto-von-Guericke University Magdeburg, Germany.
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MeSH Terms
Descriptor/Qualifier:
Adenocarcinoma / genetics,  metabolism*
Adult
Aged
Barrett Esophagus / genetics,  metabolism*
Carcinoma / genetics,  metabolism*
DNA Methylation*
DNA Modification Methylases / genetics*
DNA Repair Enzymes / genetics*
Disease Progression
Female
Gene Silencing*
Humans
Lymphatic Metastasis
Male
Metaplasia / genetics,  metabolism*
Middle Aged
Neoplasm Invasiveness
Tumor Suppressor Proteins / genetics*
Grant Support
ID/Acronym/Agency:
R01CA106176/CA/NCI NIH HHS
Chemical
Reg. No./Substance:
0/Tumor Suppressor Proteins; EC 2.1.1.-/DNA Modification Methylases; EC 2.1.1.63/MGMT protein, human; EC 6.5.1.-/DNA Repair Enzymes

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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